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Felix José Alvarez Ramires
Researcher at University of São Paulo
Publications - 55
Citations - 3072
Felix José Alvarez Ramires is an academic researcher from University of São Paulo. The author has contributed to research in topics: Heart failure & Fibrosis. The author has an hindex of 20, co-authored 48 publications receiving 2553 citations. Previous affiliations of Felix José Alvarez Ramires include University of Tennessee & University of Missouri.
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Journal ArticleDOI
Angiotensin Receptor Neprilysin Inhibition Compared With Enalapril on the Risk of Clinical Progression in Surviving Patients With Heart Failure
Milton Packer,John J.V. McMurray,Akshay S. Desai,Jianjian Gong,Martin Lefkowitz,Adel R. Rizkala,Jean L. Rouleau,Victor Shi,Scott D. Solomon,Karl Swedberg,Michael R. Zile,Karl Andersen,Juan Luis Arango,J. Malcolm O. Arnold,Jan Belohlavek,Michael Böhm,Sergey Boytsov,Lesley J. Burgess,Walter Cabrera,Carlos Calvo,Chen Huan Chen,Andrej Dukát,Yan Carlos Duarte,Andrejs Erglis,Michael Fu,Efrain Gomez,Angel Gonzàlez-Medina,Albert A. Hagège,Jun Huang,Tzventana Katova,Songsak Kiatchoosakun,Kee-Sik Kim,Ömer Kozan,Edmundo Alfredo Bayram Llamas,Felipe Martinez,Béla Merkely,Ivan Mendoza,Arend Mosterd,Marta Negrusz-Kawecka,Keijo Peuhkurinen,Felix José Alvarez Ramires,Jens Refsgaard,Arvo Rosenthal,Michele Senni,Antonio S. Sibulo,José Silva-Cardoso,Iain B. Squire,Randall C. Starling,John R. Teerlink,Johan Vanhaecke,Dragos Vinereanu,Raymond Ching-Chiew Wong +51 more
TL;DR: Angiotensin-neprilysin inhibition prevents the clinical progression of surviving patients with heart failure more effectively than angiotens in-converting enzyme inhibition.
Journal ArticleDOI
Cardiac myosin activation with omecamtiv mecarbil in systolic heart failure
John R. Teerlink,Rafael Diaz,G. Michael Felker,John J.V. McMurray,Marco Metra,Scott D. Solomon,Kirkwood F. Adams,Inder S. Anand,Alexandra Arias-Mendoza,Tor Biering-Sørensen,Michael Böhm,Diana Bonderman,John G.F. Cleland,John G.F. Cleland,Ramón Corbalán,María G. Crespo-Leiro,Ulf Dahlström,Luis E. Echeverría,James C. Fang,Gerasimos Filippatos,Cândida Fonseca,Eva Goncalvesova,Assen Goudev,Jonathan G. Howlett,David E. Lanfear,Jing Li,M. Lund,Peter S. Macdonald,Viacheslav Mareev,Shin-ichi Momomura,Eileen O'Meara,Alexander Parkhomenko,Piotr Ponikowski,Felix José Alvarez Ramires,Pranas Šerpytis,Karen Sliwa,Jindrich Spinar,Thomas M. Suter,János Tomcsányi,Hans Vandekerckhove,Dragos Vinereanu,Adriaan A. Voors,Mehmet Yilmaz,Faiez Zannad,Lucie Sharpsten,Jason C. Legg,Claire Varin,Narimon Honarpour,Siddique Abbasi,Fady I. Malik,Christopher E. Kurtz +50 more
TL;DR: Among patients with heart failure and a reduced ejection, patients who received omecamtiv mecarbil had a lower incidence of a composite of a heart-failure event or death from cardiovascular causes than those who received placebo.
Journal ArticleDOI
Angiotensin II, Transforming Growth Factor-β1and Repair in the Infarcted Heart
TL;DR: Observations suggest locally generated AngII via ATireceptor binding is correlated to TGF-β1expression and synthesis at sites of repair and remote sites in the infarcted rat heart, and the mechanism responsible for the role of AngII in TGF1 remains to be elucidated.
Journal ArticleDOI
III Diretriz Brasileira de Insuficiência Cardíaca Crônica
Edimar Alcides Bocchi,Fabiana Goulart Marcondes Braga,Silvia Moreira Ayub Ferreira,Luis Eduardo Paim Rohde,Wilson de Oliveira Jr.,Dirceu R. Almeida,Maria da Consolação Vieira Moreira,Reinaldo B. Bestetti,Solange Bordignon,Clerio F. Azevedo,Evandro Mesquita Tinoco,Ricardo Mourilhe Rocha,Victor Sarli Issa,Almir Sergio Ferraz,Fátima das Dores Cruz,Guilherme Veiga Guimarães,Vanessa dos Santos Pereira Montera,Denilson Campos de Albuquerque,Fernando Bacal,Germano Emilio Conceição Souza,João Manoel Rossi Neto,Nadine Oliveira Clausell,Silvia Marinho Martins,Alexandre Siciliano,João David de Souza Neto,Luís Felipe Moreira,Ricardo Alkmim Teixeira,Lidia Zytynski Moura,Luís Beck-da-Silva,Salvador Rassi,Estela Azeka,Estela Horowitz,Felix José Alvarez Ramires,Marcus Vinicius Simões,Renato Barroso Pereira de Castro,Vera Maria Cury Salemi,Humberto Villacorta Junior,José Henrique Vila,Ricardo Simoes,Francisco Albanesi,Marcelo Westerlund Montera +40 more
Journal ArticleDOI
Fibrosis of atria and great vessels in response to angiotensin II or aldosterone infusion
TL;DR: The appearance of atrial fibrosis and perivascular fibrosis of aorta and pulmonary artery, in rats receiving AngII or ALDO suggests these responses are not related to altered ventricular wall stress or arterial hypertension, but rather to these effector hormones of the circulating renin-angiotensin-aldosterone system.