F
Francisco J. Rivera
Researcher at Paracelsus Private Medical University of Salzburg
Publications - 63
Citations - 3264
Francisco J. Rivera is an academic researcher from Paracelsus Private Medical University of Salzburg. The author has contributed to research in topics: Neural stem cell & Progenitor cell. The author has an hindex of 27, co-authored 57 publications receiving 2609 citations. Previous affiliations of Francisco J. Rivera include University of Cambridge & University of Chile.
Papers
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Journal ArticleDOI
Applying extracellular vesicles based therapeutics in clinical trials - an ISEV position paper
Thomas Lener,Mario Gimona,Ludwig Aigner,Verena Börger,Edit I. Buzás,Giovanni Camussi,Nathalie Chaput,Devasis Chatterjee,Felipe A. Court,Hernando A. del Portillo,Lorraine O'Driscoll,Stefano Fais,Juan M. Falcón-Pérez,Ursula Felderhoff-Mueser,Lorenzo Fraile,Yong Song Gho,André Görgens,Ramesh C. Gupta,An Hendrix,Dirk M. Hermann,Andrew F. Hill,Fred H. Hochberg,Peter A. Horn,Dominique P.V. de Kleijn,Lambros Kordelas,Boris W. Kramer,Eva-Maria Krämer-Albers,Sandra Laner-Plamberger,Saara Laitinen,Tommaso Leonardi,Magdalena J. Lorenowicz,Sai Kiang Lim,Jan Lötvall,Casey A. Maguire,Antonio Marcilla,Irina Nazarenko,Takahiro Ochiya,Tushar Patel,Shona Pedersen,Gabriella Pocsfalvi,Stefano Pluchino,Peter J. Quesenberry,Ilona G. Reischl,Francisco J. Rivera,Ralf Sanzenbacher,Katharina Schallmoser,Ineke Slaper-Cortenbach,Dirk Strunk,Torsten Tonn,Pieter Vader,Pieter Vader,Bas W.M. van Balkom,Marca H. M. Wauben,Samir El Andaloussi,Samir El Andaloussi,Clotilde Théry,Eva Rohde,Bernd Giebel +57 more
TL;DR: In this paper, the authors summarize recent developments and the current knowledge of extracellular vesicles (EVs) and discuss safety and regulatory requirements that must be considered for pharmaceutical manufacturing and clinical application.
Journal ArticleDOI
Brain and Retinal Pericytes: Origin, Function and Role
Andrea Trost,Simona Lange,Falk Schroedl,Daniela Bruckner,Karolina Motloch,Barbara Bogner,Alexandra Kaser-Eichberger,Clemens Strohmaier,Christian Runge,Ludwig Aigner,Francisco J. Rivera,Herbert A. Reitsamer +11 more
TL;DR: An overview of the current understanding of CNS pericyte origin and function is provided, with a special focus on the retina in the healthy and diseased, and the role of pericytes in de- and regenerative processes is highlighted.
Journal ArticleDOI
Mesenchymal Stem Cells Instruct Oligodendrogenic Fate Decision on Adult Neural Stem Cells
Francisco J. Rivera,Francisco J. Rivera,Sebastien Couillard-Despres,Xiomara Pedré,Sonja Ploetz,Massimiliano Caioni,Carlos Lois,Ulrich Bogdahn,Ludwig Aigner +8 more
TL;DR: It is revealed that the interactions between adult MSCs and NSCs, mediated by soluble factors, induce oligodendrogenic fate decision in N SCs at the expense of astrogenesis.
Journal ArticleDOI
Dynamic of distribution of human bone marrow-derived mesenchymal stem cells after transplantation into adult unconditioned mice
Carolina Allers,Walter Sierralta,Sonia Neubauer,Francisco J. Rivera,José J. Minguell,Paulette Conget +5 more
TL;DR: The results suggest that human MSC transplantation into unconditioned recipients represents an option for providing cellular therapy and avoids the complications associated with drugs or radiation conditioning.
Journal ArticleDOI
Structural and functional rejuvenation of the aged brain by an approved anti-asthmatic drug
Julia Marschallinger,Iris Schäffner,Barbara Klein,Renate Gelfert,Francisco J. Rivera,Sebastian Illes,Lukas Grassner,Maximilian Janssen,Peter Rotheneichner,Claudia Schmuckermair,Roland Coras,Marta Boccazzi,Mansoor Chishty,Florian B. Lagler,Marija Renic,Hans-Christian Bauer,Nicolas Singewald,Ingmar Blümcke,Ulrich Bogdahn,Sebastien Couillard-Despres,D. Chichung Lie,Maria P. Abbracchio,Ludwig Aigner +22 more
TL;DR: This work shows that a 6-week treatment of young and old rats with montelukast, a marketed anti-asthmatic drug antagonizing leukotriene receptors, reduces neuroinflammation, elevates hippocampal neurogenesis and improves learning and memory in old animals, and demonstrates that the effect is mediated through inhibition of the GPR17 receptor.