M
Maja Krajinovic
Researcher at Université de Montréal
Publications - 162
Citations - 4606
Maja Krajinovic is an academic researcher from Université de Montréal. The author has contributed to research in topics: Childhood Acute Lymphoblastic Leukemia & Population. The author has an hindex of 34, co-authored 143 publications receiving 4041 citations. Previous affiliations of Maja Krajinovic include Centre Hospitalier Universitaire Sainte-Justine & International Centre for Genetic Engineering and Biotechnology.
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Journal ArticleDOI
Variation in CDKN2A at 9p21.3 influences childhood acute lymphoblastic leukemia risk
Amy L. Sherborne,Fay J. Hosking,Rashmi B. Prasad,Rajesh Kumar,Rolf Koehler,Jayaram Vijayakrishnan,Elli Papaemmanuil,Claus R. Bartram,Martin Stanulla,Martin Schrappe,Andreas Gast,Sara E. Dobbins,Yussanne Ma,Eamonn Sheridan,Malcolm Taylor,Sally E. Kinsey,T Lightfoot,Eve Roman,Julie Irving,James M. Allan,Anthony V. Moorman,Christine J. Harrison,Ian Tomlinson,Sue Richards,Martin Zimmermann,Csaba Szalai,Ágnes F. Semsei,Dániel J. Erdélyi,Maja Krajinovic,Daniel Sinnett,Jasmine Healy,Anna Gonzalez Neira,Norihiko Kawamata,Seishi Ogawa,H. Phillip Koeffler,Kari Hemminki,Mel Greaves,Richard S. Houlston +37 more
TL;DR: Common variation at 9p21.3 (rs3731217, intron 1 of CDKN2A) influences acute lymphoblastic leukemia risk (odds ratio = 0.71, P = 3.01 × 10−11), irrespective of cell lineage.
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Susceptibility to Childhood Acute Lymphoblastic Leukemia: Influence of CYP1A1, CYP2D6, GSTM1, and GSTT1 Genetic Polymorphisms
TL;DR: The results suggest that the risk of ALL may indeed be associated with xenobiotics-metabolism, and thus with environmental exposures, and may also explain, in part, why ALL is more prevalent among males than females.
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Multidrug resistance gene (MDR1) polymorphisms are associated with major molecular responses to standard-dose imatinib in chronic myeloid leukemia
Stéphanie Dulucq,Stéphane Bouchet,Béatrice Turcq,Eric Lippert,Gabriel Etienne,Josy Reiffers,Mathieu Molimard,Maja Krajinovic,Francois-Xavier Mahon +8 more
TL;DR: The usefulness of these single nucleotide polymorphisms of MDR1 in the identification of CML who may or may not respond optimally to imatinib is demonstrated.
Journal ArticleDOI
A new locus for arrhythmogenic right ventricular dysplasia on the long arm of chromosome 14
Giovanni Maria Severini,Maja Krajinovic,Bruno Pinamonti,Gianfranco Sinagra,Paolo Fioretti,Maria Cristiana Brunazzi,Arturo Falaschi,Fulvio Camerini,Mauro Giacca,Luisa Mestroni,Luisa Mestroni +10 more
TL;DR: Data indicate that a novel gene causing familial ARVD (provisionally named ARVD2) maps to the long arm of chromosome 14, thus supporting the hypothesis of genetic heterogeneity in this disease.
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Association of busulfan exposure with survival and toxicity after haemopoietic cell transplantation in children and young adults: a multicentre, retrospective cohort analysis
Imke H. Bartelink,Arief Lalmohamed,Elisabeth M L van Reij,Christopher C. Dvorak,Rada M. Savic,Juliette Zwaveling,Robbert G. M. Bredius,Antoine C. G. Egberts,Marc Bierings,Morris Kletzel,Peter J. Shaw,Christa E. Nath,George Hempel,Marc Ansari,Maja Krajinovic,Yves Théorêt,Yves Théorêt,Michel Duval,Ron J. Keizer,Henrique Bittencourt,Moustapha Hassan,Tayfun Güngör,Robert Wynn,Paul Veys,Geoff D.E. Cuvelier,Sarah Marktel,Roberto Chiesa,Morton J. Cowan,Mary Slatter,Melisa K. Stricherz,Cathryn Jennissen,Janel Long-Boyle,Jaap Jan Boelens +32 more
TL;DR: The main outcome of interest was event-free survival and improved clinical outcomes are likely to be achieved by targeting the busulfan AUC to 78-101 mg hokinetic model for all indications.