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Nora D. Volkow

Researcher at National Institute on Drug Abuse

Publications -  1038
Citations -  121498

Nora D. Volkow is an academic researcher from National Institute on Drug Abuse. The author has contributed to research in topics: Dopamine & Addiction. The author has an hindex of 165, co-authored 958 publications receiving 107463 citations. Previous affiliations of Nora D. Volkow include National Institutes of Health & North Shore University Hospital.

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PET imaging in clinical drug abuse research.

TL;DR: PET and SPECT have been used to help the understanding of many aspects of the pharmacokinetics and pharmacodynamics of abused drugs, and have made valuable contributions in terms of drug mechanisms, drug interactions, and drug toxicities.
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Bariatric surgery in obese patients reduced resting connectivity of brain regions involved with self-referential processing.

TL;DR: Bariatric surgery significantly decreased FCD in regions involved in self‐referential processing (VMPFC, D MPFC, dACC, and precuneus), and interoception (insula), and changes in VMPFC/precuneus were associated with reduction in BMI suggesting a role in improving control of eating behaviors following surgery.
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Ultrasensitive detection of 3D cerebral microvascular network dynamics in vivo

TL;DR: This paper reports on a new method, contrast-enhanced ODT with Intralipid that significantly improves quantitative CBFv imaging of capillary networks by obviating the errors from long latency between flowing red blood cells (low hematocrit ~20% in capillaries).
Journal Article

Effects of Electroconvulsive Therapy on Brain Glucose Metabolism: A Preliminary Study.

TL;DR: Depressed individuals showed decreased uptake of FDG in the frontal cortex after ECT treatment and studies of radioactive uptake were done using FDG as a measure for glucose metabolism and with oxygen-15-labeled water as aMeasure for cerebral blood flow.
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Leptin increases striatal dopamine D2 receptor binding in leptin‐deficient obese (ob/ob) mice

TL;DR: Findings provide further evidence that leptin modulates D2R expression in striatum and that these effects are genotype/phenotype dependent, and suggest that leptin‐deficient mice show increased DA activity in reward‐related brain regions.