R
Ronald Klein
Researcher at University of Wisconsin-Madison
Publications - 1306
Citations - 163459
Ronald Klein is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Population & Diabetes mellitus. The author has an hindex of 194, co-authored 1305 publications receiving 149140 citations. Previous affiliations of Ronald Klein include Los Angeles Biomedical Research Institute & Wake Forest University.
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Journal Article
Does Refractive Error Influence the Association of Blood Pressure and Retinal Vessel Diameters? The Blue Mountains Eye Study
Ava Grace Tan,Tien Yin Wong,Jie Jin Wang,Elena Rochtchina,Ronald Klein,Paul Mitchell,Blue Mountains Eye Study +6 more
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Diabetes, hyperglycemia, and age-related maculopathy. The Beaver Dam Eye Study.
TL;DR: In this paper, the authors examined the association among hyperglycemia, diabetes status, and age-related maculopathy in a population-based study of people between the ages of 43 and 86 years who lived in Beaver Dam, Wisconsin between 1988 and 1990.
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An Approach to the Prevention of Blindness in Diabetes
TL;DR: This approach requires that diabetic patients who are at high risk for visual loss, and not under the care of ophthalmologists, be systematically screened, referred, and treated.
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Reproductive exposures, incident age-related cataracts, and age-related maculopathy in women: the Beaver Dam Eye Study
TL;DR: In this paper, the authors evaluated the relationship of reproductive exposures and incident age-related cataract and maculopathy in women and found no significant associations of any reproductive exposure with lesions of early or late age related maculopathies.
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A spirulina-enhanced diet provides neuroprotection in an α-synuclein model of Parkinson's disease
Mibel Pabon,Jennifer Jernberg,Josh M. Morganti,Jessika Contreras,Charles Hudson,Ronald Klein,Paula C. Bickford,Paula C. Bickford +7 more
TL;DR: Spirulina was neuroprotective in this α-synuclein model of PD as more TH+ and NeuN+ cells were observed and spirulina concomitantly decreased the numbers of activated microglial cells as determined by MHCII expression.