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Nucleotide Polymerase Inhibitor So fos bu vir plus Ribavirin for Hepatitis C

TLDR
Sofosbuvir plus ribavirin for 12 weeks may be effective in previously untreated patients with HCV genotype 1, 2, or 3 infection, and the rate of sustained virologic response 24 weeks after therapy is reported.
Abstract
BACKGROUND The standard treatment for hepatitis C virus (HCV) infection is interferon, which is administered subcutaneously and can have troublesome side effects. We evaluated so fos bu vir, an oral nucleotide inhibitor of HCV polymerase, in interferon-sparing and interferon-free regimens for the treatment of HCV infection. METHODS

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DNA polymerase θ specializes in incorporating synthetic expanded-size (xDNA) nucleotides

TL;DR: A unique function for Polθ is demonstrated in incorporating synthetic large-sized nucleotides into DNA large benzo-expanded nucleotide analogs which exhibit canonical base-pairing and enhanced base stacking and suggest the future possibility of the use of dxG nucleoside or related prodrug analogs as selective inhibitors of Polη activity.
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Management of acute and chronic HCV infection in persons with HIV coinfection.

TL;DR: The anticipated availability of safe, tolerable and highly efficacious interferon-free, oral HCV direct-acting antiviral combination therapies promise to dramatically change the management of acute and chronic HCV infection in HIV-infected persons.
Journal ArticleDOI

The Role of Chemokines in Hepatitis C Virus-Mediated Liver Disease

TL;DR: HCV-mediated changes in hepatic immune cell chemotaxis during the chronic phase of the infection are significantly affecting antiviral immunity and tissue damage and thus influence survival of both the host and the virus.
Journal ArticleDOI

Sofosbuvir-based interferon-free therapy for patients with HCV infection

TL;DR: Sofosbuvir plus ribavirin for 12 weeks may be effective in previously untreated patients with HCV genotype 1, 2, or 3 infec- tion, and the rate of sustained virologic response 24 weeks after therapy is reported.
References
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Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance.

TL;DR: It is reported that a genetic polymorphism near the IL28B gene, encoding interferon-λ-3 (IFN-α-2a) is associated with an approximately twofold change in response to treatment, both among patients of European ancestry and African-Americans.
Journal ArticleDOI

Diagnosis, management, and treatment of hepatitis C: An update

TL;DR: This document has been approved by the AASLD, the Infectious Diseases Society of America, and the American College of Gastroenterology.
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Discovery of a β-d-2'-deoxy-2'-α-fluoro-2'-β-C-methyluridine nucleotide prodrug (PSI-7977) for the treatment of hepatitis C virus.

TL;DR: Phosphoramidate prodrugs of the 5'-phosphate derivative of the β-d- 2'-deoxy-2'-α-fluoro-2-β-C-methyluridine nucleoside showed significant potency in the HCV subgenomic replicon assay and produced high levels of triphosphates 6 in primary hepatocytes and in the livers of rats, dogs, and monkeys when administered in vivo.
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Preliminary Study of Two Antiviral Agents for Hepatitis C Genotype 1

TL;DR: This preliminary study involving patients with chronic HCV genotype 1 infection who had not had a response to prior therapy showed that a sustained virologic response can be achieved with two direct-acting antiviral agents only.
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Naturally occurring dominant resistance mutations to hepatitis c virus protease and polymerase inhibitors in treatment-naive patients

TL;DR: Naturally occurring dominant STAT‐C resistance mutations are common in treatment‐naïve patients infected with HCV genotype 1, and their influence on treatment outcome should be characterized to evaluate possible benefits of drug resistance testing for individual tailoring of drug combinations when treatment options are limited due to previous nonresponse to peginterferon and ribavirin.
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