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Bradley N. Jack

Researcher at Australian National University

Publications -  35
Citations -  425

Bradley N. Jack is an academic researcher from Australian National University. The author has contributed to research in topics: Binocular rivalry & Medicine. The author has an hindex of 9, co-authored 28 publications receiving 266 citations. Previous affiliations of Bradley N. Jack include Leipzig University & Southern Cross University.

Papers
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Neurophysiological evidence of efference copies to inner speech

TL;DR: The results demonstrate that inner speech – a purely mental action – is associated with an efference copy with detailed auditory properties, and suggest that innerspeech may ultimately reflect a special type of overt speech.
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The neurophenomenology of early psychosis: An integrative empirical study.

TL;DR: A neurophenomenological model of minimal self-disturbance in schizophrenia spectrum disorders may need to be expanded from source monitoring deficits to encompass other relevant constructs such as temporal processing, intermodal/multisensory integration, and hierarchical predictive processing.
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Goal-Directed and Habit-Like Modulations of Stimulus Processing during Reinforcement Learning.

TL;DR: The results demonstrate for the first time that visual cortex activity is increased for reward-related stimuli even when the rewarding event is temporarily devalued, and shows that both habit-like attention and goal-directed processes occur in the same learning episode at different latencies.
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#EEGManyLabs : investigating the replicability of influential EEG experiments

Yuri G. Pavlov, +58 more
- 02 Apr 2021 - 
TL;DR: The #EEGManyLabs project as discussed by the authors is a large-scale international collaborative replication effort that aims to evaluate the replicability of EEG findings about the relationship between brain activity and cognitive phenomena.
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Self‐initiated actions result in suppressed auditory but amplified visual evoked components in healthy participants

TL;DR: A difference in sensory processing of self-initiated stimuli across modalities is highlighted, and may have implications for clinical disorders that are ostensibly associated with abnormal self-suppression.