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Chris J.L.M. Meijer

Researcher at VU University Amsterdam

Publications -  745
Citations -  83366

Chris J.L.M. Meijer is an academic researcher from VU University Amsterdam. The author has contributed to research in topics: Cervical cancer & Cervical intraepithelial neoplasia. The author has an hindex of 128, co-authored 733 publications receiving 78705 citations. Previous affiliations of Chris J.L.M. Meijer include VU University Medical Center & Academic Center for Dentistry Amsterdam.

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Male circumcision, penile human papillomavirus infection, and cervical cancer in female partners

TL;DR: Empirical evidence is provided that male circumcision lowers the personal risk of genital HPV infection and seems to lower the chance of cervical cancer developing in women who have high-risk sex partners.
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Human papillomavirus—the most significant risk determinant of cervical intraepithelial neoplasia

TL;DR: Cervical HPV infection was by far the most significant risk factor for cervical squamous intraepithelial lesions and the importance of the previously identified epidemiological risk factors for cervical neoplasia was also demonstrated.
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Risk factors for genital HPV DNA in men resemble those found in women: a study of male attendees at a Danish STD clinic.

TL;DR: Most risk factors for HPV DNA detection in men resemble those found in women, including whether there are different risk profiles for oncogenic and non-oncogenic HPV types.
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Addition of high-risk HPV testing improves the current guidelines on follow-up after treatment for cervical intraepithelial neoplasia

TL;DR: Largely overlapping, partly different groups of women with post-treatment CIN 2/3 were identified by HPV testing and cervical cytology and it is advocated to include high-risk HPV testing in monitoring women initially treated for CIN2/3.
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p53 expression above the basal cell layer in oral mucosa is an early event of malignant transformation and has predictive value for developing oral squamous cell carcinoma

TL;DR: The results suggest that clear expression of p53 above the basal cell layer is an early event in oral carcinogenesis and an indicator of a developing carcinoma, even preceding morphological tissue alterations.