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Chris J.L.M. Meijer

Researcher at VU University Amsterdam

Publications -  745
Citations -  83366

Chris J.L.M. Meijer is an academic researcher from VU University Amsterdam. The author has contributed to research in topics: Cervical cancer & Cervical intraepithelial neoplasia. The author has an hindex of 128, co-authored 733 publications receiving 78705 citations. Previous affiliations of Chris J.L.M. Meijer include VU University Medical Center & Academic Center for Dentistry Amsterdam.

Papers
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The health and economic effects of HPV DNA screening in The Netherlands

TL;DR: The model indicates that HPV testing with cytology triage is likely to be cost effective and an extension of the screening interval may be considered to control costs.
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EORTC classification for primary cutaneous lymphomas: the best guide to good clinical management. European Organization for Research and Treatment of Cancer.

TL;DR: In 1997, the Cutaneous Lymphoma Study Group of the European Organization for Research and Treatment of Cancer (EORTC) published a proposal for a classification for the group of primary cutaneous lymphomas (1).
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Classification of primary cutaneous T-cell lymphomas.

TL;DR: A new classification of cutaneous T‐cell lymphoma is presented, based on a combination of clinical, histological and immunophenotypic criteria and it recognizes distinct clinico‐pathological entities within this group of diseases.
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Telomerase suppression by chromosome 6 in a human papillomavirus type 16-immortalized keratinocyte cell line and in a cervical cancer cell line

TL;DR: Ectopic expression of hTERT in FK16A cells could prevent the telomeric shortening-based growth arrest induced by chromosome 6, and Chromosome 6 may harbor a repressor of h TERT transcription, the loss of which may be involved in HPV-mediated immortalization.
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Integrated genomic and transcriptional profiling identifies chromosomal loci with altered gene expression in cervical cancer.

TL;DR: Integrated chromosomal and transcriptional profiling identified chromosomal hotspots at 1q, 3q, 11q, and 20q with altered gene expression within large commonly altered chromosomal regions in cervical cancer.