D
David A. Jackson
Researcher at King's College London
Publications - 1166
Citations - 76015
David A. Jackson is an academic researcher from King's College London. The author has contributed to research in topics: Optical fiber & Interferometry. The author has an hindex of 136, co-authored 1095 publications receiving 68352 citations. Previous affiliations of David A. Jackson include University of California, Berkeley & University of Alberta.
Papers
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Strain sensing of modern composite materials with a spatial/wavelength-division multiplexed fiber grating network
TL;DR: A spatial/wavelength-division multiplexing topology with combination of a tunable-wavelength filter and an interferometric wavelength scanner is used to interrogate a range of fiber Bragg grating (FBG) strain sensors embedded in modern composite materials.
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A fluorometric assay for trehalose in the picomole range.
Petronia Carillo,Regina Feil,Yves Gibon,Namiko Satoh-Nagasawa,David A. Jackson,Oliver Bläsing,Mark Stitt,John E. Lunn +7 more
TL;DR: In this paper, Trehalose 6-phosphate (Tre6P), the intermediate of trehalose biosynthesis, is thought to be a signal of sucrose status.
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Demodulation scheme fibre interferometric sensors employing laser frequency switching
TL;DR: In this paper, a demodulation technique suitable for use with fiber interferometric sensors is described, which employs a frequency switched diode laser and an unbalanced Mach-Zehnder interferometer.
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Lymphatic exosomes promote dendritic cell migration along guidance cues.
Markus Brown,Louise A. Johnson,Dario A. Leone,Peter Májek,Kari Vaahtomeri,Daniel Senfter,Nora Bukosza,Helga Schachner,Gabriele Asfour,Brigitte Langer,Robert Hauschild,Katja Parapatics,Young-Kwon Hong,Keiryn L. Bennett,Renate Kain,Michael Detmar,Michael Sixt,David A. Jackson,Dontscho Kerjaschki +18 more
TL;DR: It is concluded that perilymphatic LEC exosomes enhance exploratory behavior and thus promote directional migration of CX3CR1-expressing cells in complex tissue environments.
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A Mechanism of Sialylation Functionally Silences the Hyaluronan Receptor LYVE-1 in Lymphatic Endothelium
TL;DR: In this article, the major lymphatic endothelial hyaluronan receptor LYVE-1 is functionally "silenced" in a cell-specific fashion by autoinhibitory glycosylation.