J
James T. Rutka
Researcher at University of Toronto
Publications - 642
Citations - 34051
James T. Rutka is an academic researcher from University of Toronto. The author has contributed to research in topics: Epilepsy & Epilepsy surgery. The author has an hindex of 84, co-authored 618 publications receiving 30391 citations. Previous affiliations of James T. Rutka include Princeton University & Toronto Western Hospital.
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"Polyphenotypic" tumors in the central nervous system: problems in nosology and classification.
TL;DR: Although the polyphenotype raises the issue that these may represent variants of MRT or the atypical teratoid-rhabdoid tumor, the morphologic findings in the two cases were very dissimilar, underscore the problems in nosology and classification of polyphenotypic tumors of the CNS.
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Meningioma with dural venous sinus invasion and jugular vein extension.
TL;DR: The case of an intracranial meningioma with invasion of, and intraluminal extension into, the transverse and sigmoid sinuses, jugular bulb, and internal jugular vein, resulting in venous occlusion in a 14-year-old girl.
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Magnetoencephalographic spike sources associated with auditory auras in paediatric localisation-related epilepsy.
Ismail S. Mohamed,Hiroshi Otsubo,Elizabeth W. Pang,S. H. Chuang,James T. Rutka,P Dirks,Shelly K. Weiss,O. C. Snead +7 more
TL;DR: MEG spike sources clustered in the superior temporal gyrus in six patients with auditory auras, which contained the residual or recurrent epileptogenic zone after incomplete cortical excision for lesional epilepsy.
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A functional genomics approach to identify pathways of drug resistance in medulloblastoma.
Kelsey C. Bertrand,Kelsey C. Bertrand,Claudia C. Faria,Patryk Skowron,Amanda Luck,Livia Garzia,Xiaochong Wu,Sameer Agnihotri,Christian A. Smith,Michael D. Taylor,Stephen C. Mack,Stephen C. Mack,James T. Rutka +12 more
TL;DR: A spontaneous metastatic mouse model of medulloblastoma driven by the Sleeping Beauty mutagenesis transposon system serves as a novel approach to dissecting patterns of therapy resistance at multiple tumor sites simultaneously which can be readily applied to other cancer systems.
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Changing ictal-onset EEG patterns in children with cortical dysplasia.
Elaine Galicia,Katsumi Imai,Ismail S. Mohamed,Cristina Go,Ayataka Fujimoto,Ayako Ochi,Ryoichi Sakuta,William Halliday,James T. Rutka,Sylvester H. Chuang,O. Carter Snead,Hiroshi Otsubo +11 more
TL;DR: Ictal-onset EEG patterns change over time in children with early seizure onset and intractable epilepsy caused by CD, and younger epileptic children with CD more frequently have multifocal epileptic foci or larger epileptogenic foci.