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Jon Lindstrom

Researcher at University of Pennsylvania

Publications -  442
Citations -  50369

Jon Lindstrom is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Acetylcholine receptor & Nicotinic agonist. The author has an hindex of 108, co-authored 441 publications receiving 48999 citations. Previous affiliations of Jon Lindstrom include University of California, San Diego & University of California, Riverside.

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Expression of cholinergic system molecules during development of the chick nervous system.

TL;DR: A developmentally regulated expression of cholinergic system-related molecules in the chick nervous system is revealed and differential time-courses of expression for nAChR subunits, AChE, and ChAT during development are characterized.
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Distinctive effects of nicotinic receptor intracellular-loop mutations associated with nocturnal frontal lobe epilepsy.

TL;DR: Both common and differing features between TM- and C2-domain AD/NFLE-associated mutations are identified, and shared features may be particularly salient to AD/ NFLE etiology.
Journal Article

Myasthenia gravis--current concepts.

TL;DR: Current findings indicate that autoimmune myasthenia gravis is an acquired immune complex disorder of neuromuscular transmission in voluntary striated muscle, and anticholinesterases, corticosteroids, immunosuppressants, plasmapheresis or thymectomy (individually or in combination) provide control and better prognosis in most patients.
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Identification of cholinoceptive glycinergic neurons in the mammalian retina.

TL;DR: It is demonstrated that about 70% of the cholinoceptive amacrine cells in rabbit retina are glycinergic cells, which indicates that ACh in the mammalian retina may modulate Glycinergic circuits via extrasynaptic β2α3 nAChRs.
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Common variants in beta2- and beta3-adrenergic receptor genes and uncoupling protein 1 as predictors of the risk for type 2 diabetes and body weight changes. The Finnish Diabetes Prevention Study.

TL;DR: No significant association was observed between the three polymorphisms and anthropometric measurements, glucose and insulin levels, after correcting for multiple testing, and the risk of T2DM did not differ among the three genotypes of the b2-AR gene.