J
Jon Lindstrom
Researcher at University of Pennsylvania
Publications - 442
Citations - 50369
Jon Lindstrom is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Acetylcholine receptor & Nicotinic agonist. The author has an hindex of 108, co-authored 441 publications receiving 48999 citations. Previous affiliations of Jon Lindstrom include University of California, San Diego & University of California, Riverside.
Papers
More filters
Journal ArticleDOI
Plasma insulin and cardiovascular mortality in non-diabetic European men and women: a meta-analysis of data from eleven prospective studies.
Aulikki Nissinen,Juha Pekkanen,Jaakko Tuomilehto,P. Jousilahti,Jon Lindstrom,M. Pyorala,Kalevi Pyörälä,G. Gallus,M.P. Garancini,Lex M. Bouter,J.M. Dekker,R.J. Heine,Giel Nijpels,Coen D.A. Stehouwer,E. J. M. Feskens,Daan Kromhout,Andrzej Pajak,Mats Eliasson,Bernd Stegmayr,V. Lundberg,Nicholas J. Wareham,Nigel Unwin,N. Ahmad,K. G. M. M. Alberti,L. Hayes,John S Yudkin,M.M. Gould,Andy Haines,Richard W Morris,Knut Borch-Johnsen,Johan G. Eriksson,Qing Qiao,Gang Hu +32 more
TL;DR: Hyperinsulinaemia, defined by the highest quartile cut-off for fasting insulin, was significantly associated with cardiovascular mortality in both men and women independently of other risk factors, and associations between high 2-h insulin and cardiovascular mortality were weaker and non-significant.
Journal ArticleDOI
Content of acetylcholine receptor and antibodies bound to receptor in myasthenia gravis, experimental autoimmune myasthenia gravis, and Eaton-Lambert syndrome.
Jon Lindstrom,Edward H. Lambert +1 more
TL;DR: In both myasthenia gravis and its animal model, a decrease in the content of fully active receptor is probably the most important factor impairing neuromuscular transmission.
Book ChapterDOI
Autoimmune Response to Acetylcholine Receptors in Myasthenia Gravis and Its Animal Model
TL;DR: This chapter discusses the process of neurotransmission as well as acetylcholine receptors (AChR) structure and function to understand the pathological mechanisms involved in myasthenia gravis (MG) and experimental autoimmune myASThenia Gravis (EAMG).
Journal ArticleDOI
The motor end plate in myasthenia gravis and in experimental autoimmune myasthenia gravis. A quantitative ultrastructural study.
TL;DR: The postsynaptic region is the primary target of the autoimmune reaction in EAMG and the ultrastructural and morphometric abnormalities of the end plate in the chronic phase of the syndrome closely resemble those which have been observed in human myasthenia gravis.