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Betty P. Tsao

Researcher at Medical University of South Carolina

Publications -  49
Citations -  6582

Betty P. Tsao is an academic researcher from Medical University of South Carolina. The author has contributed to research in topics: Lupus erythematosus & Single-nucleotide polymorphism. The author has an hindex of 29, co-authored 49 publications receiving 5249 citations. Previous affiliations of Betty P. Tsao include University of California, Los Angeles.

Papers
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Journal ArticleDOI

Human polymorphism at microRNAs and microRNA target sites.

Liuqing Yang, +168 more
- 01 Jan 2013 - 
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Genome-wide association scan in women with systemic lupus erythematosus identifies susceptibility variants in ITGAM, PXK, KIAA1542 and other loci.

John B. Harley, +46 more
- 20 Jan 2008 - 
TL;DR: The results show that numerous genes, some with known immune-related functions, predispose to SLE, and evidence of association with replication is found at FCGR2A, PTPN22 and STAT4, regions previously associated with SLE and other autoimmune diseases.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Association of a functional variant downstream of TNFAIP3 with systemic lupus erythematosus

Indra Adrianto, +54 more
- 01 Mar 2011 - 
TL;DR: These results establish this TT>A variant as the most likely functional polymorphism responsible for the association between TNFAIP3 and SLE.
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Evaluation of the TREX1 gene in a large multi-ancestral lupus cohort

Bahram Namjou, +42 more
- 01 Jun 2011 - 
TL;DR: Analysis of common TREX1 SNPs revealed a relatively common risk haplotype in European SLE patients with neurological manifestations, especially seizures, with a frequency of 58% in lupus cases compared with 45% in normal controls, and a strong association with anti-nRNP was observed.