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Mahmut Tör

Researcher at University of Worcester

Publications -  73
Citations -  6217

Mahmut Tör is an academic researcher from University of Worcester. The author has contributed to research in topics: Gene & Arabidopsis. The author has an hindex of 27, co-authored 64 publications receiving 5422 citations. Previous affiliations of Mahmut Tör include Warwick HRI & University of Warwick.

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A naturally occurring epigenetic mutation in a gene encoding an SBP-box transcription factor inhibits tomato fruit ripening.

TL;DR: It is demonstrated that an SBP-box gene is critical for normal ripening and highlights the likely importance of epialleles in plant development and the generation of natural variation.
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The Arabidopsis Leucine-Rich Repeat Receptor–Like Kinases BAK1/SERK3 and BKK1/SERK4 Are Required for Innate Immunity to Hemibiotrophic and Biotrophic Pathogens

TL;DR: This work demonstrates that the leucine-rich receptor kinases EFR and FLS2 form a ligand-induced complex with several LRR-RKs that belong to the SERK subfamily, and reveals that BAK1 and BKK1 contribute to disease resistance against the hemibiotrophic bacterium Pseudomonas syringae and the obligate biotrophic oomycete Hyaloperonospora arabidopsidis.
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The grateful dead: damage-associated molecular pattern molecules and reduction/oxidation regulate immunity.

TL;DR: It is speculated that their destruction through oxidative mechanisms normally exerted by myeloid cells, such as neutrophils and eosinophils, or their persistence in the setting of pathologic extracellular reducing environments, maintained by exuberant necrotic cell death and/or oxidoreductases, represent important molecular means enabling chronic inflammatory states.
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The Top 10 oomycete pathogens in molecular plant pathology

TL;DR: A survey to query the community for their ranking of plant-pathogenic oomycete species based on scientific and economic importance received 263 votes from 62 scientists in 15 countries for a total of 33 species and the Top 10 species are provided.
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Timing of plant immune responses by a central circadian regulator.

TL;DR: Surprisingly, it is found that these new defence genes are under circadian control by CCA1, allowing plants to ‘anticipate’ infection at dawn when the pathogen normally disperses the spores and time immune responses according to the perception of different pathogenic signals upon infection.