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Michael Detmar

Researcher at École Polytechnique Fédérale de Lausanne

Publications -  351
Citations -  43193

Michael Detmar is an academic researcher from École Polytechnique Fédérale de Lausanne. The author has contributed to research in topics: Lymphatic system & Lymphangiogenesis. The author has an hindex of 94, co-authored 334 publications receiving 39086 citations. Previous affiliations of Michael Detmar include Harvard University & Beth Israel Deaconess Medical Center.

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Journal ArticleDOI

Tumor Lymphangiogenesis Predicts Melanoma Metastasis to Sentinel Lymph Nodes

TL;DR: Assessment of lymphangiogenesis in primary melanomas may be a more effective approach than measuring tumor thickness in selecting patients with early metastatic disease for aggressive therapy.
Journal Article

Initial hyperproliferation and incomplete terminal differentiation of cultured human keratinocytes from lesional and uninvolved psoriatic skin.

TL;DR: The results indicate that the characteristic hyperproliferation of psoriatic KCs may only be maintained over a short period of primary culture, whereas defective terminal differentiation of lesional psoriasis KCs was maintained throughout the culture period.
Journal ArticleDOI

Präemyopathische versus amyopathische Dermatomyositis

TL;DR: In this paper, the authors describe two cases and discuss the differential diagnosis and therapy; also, the term "Premyopathic dermatomyositis" is proposed, to indicate that the full picture is to be expected in most cases.
Book ChapterDOI

Visualization and Measurement of Lymphatic Function In Vivo.

TL;DR: Three protocols for assays based upon near-infrared fluorescence imaging that can visualize and quantify different aspects of lymphatic function in mice are provided.
Journal ArticleDOI

Characterization of Tumor Blood Vasculature Expression of Human Invasive Bladder Cancer by Laser Capture Microdissection and Transcriptional Profiling.

TL;DR: The results indicate that ANGPTL2 might serve as a potential biomarker to predict progression-free survival in I-BLCA, and semi-quantitively analyzed the expression of ANG PTL2 in tissue microarrays from I-bLCA and found surprisingly an opposite correlation between staining intensity and progression- free survival in the I- BLCA.