scispace - formally typeset
M

Mei-Hua Hall

Researcher at Harvard University

Publications -  90
Citations -  3893

Mei-Hua Hall is an academic researcher from Harvard University. The author has contributed to research in topics: Psychosis & Bipolar disorder. The author has an hindex of 31, co-authored 81 publications receiving 3194 citations. Previous affiliations of Mei-Hua Hall include University of London & McLean Hospital.

Papers
More filters
Journal ArticleDOI

Genomic Dissection of Bipolar Disorder and Schizophrenia, Including 28 Subphenotypes

Douglas M. Ruderfer, +631 more
- 14 Jun 2018 - 
TL;DR: For the first time, specific loci that distinguish between BD and SCZ are discovered and polygenic components underlying multiple symptom dimensions are identified that point to the utility of genetics to inform symptomology and potential treatment.
Journal ArticleDOI

Rare coding variants in ten genes confer substantial risk for schizophrenia

Tarjinder Singh, +110 more
- 08 Apr 2022 - 
TL;DR: In this paper , a meta-analysing the whole exomes of 24,248 schizophrenia cases and 97,322 controls was used to implicate ultra-rare coding variants in 10 genes as conferring substantial risk for schizophrenia (odds ratios of 3-50, P < 2.14 × 10-6) and 32 genes at a false discovery rate of < 5%.
Journal ArticleDOI

Varied effects of atypical neuroleptics on P50 auditory gating in schizophrenia patients

TL;DR: Improvement in P50 gating appears to be greatest in patients treated with clozapine, and this study sought to determine whether other atypical neuroleptics improve P50 ratios.
Journal ArticleDOI

Heritability and Reliability of P300, P50 and Duration Mismatch Negativity

TL;DR: The high reliability and heritability of the P300 amplitude, MMN amplitude, and P50 suppression ratio components supports their use as candidate endophenotypes for psychiatric research.
Journal ArticleDOI

White matter microstructural impairments and genetic liability to familial bipolar I disorder

TL;DR: Increased genetic liability for bipolar disorder was associated with reduced fractional anisotropy across distributed regions of white matter in patients and their unaffected relatives.