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Scott M. Grundy

Researcher at University of Texas Southwestern Medical Center

Publications -  849
Citations -  246629

Scott M. Grundy is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Cholesterol & Lipoprotein. The author has an hindex of 187, co-authored 841 publications receiving 231821 citations. Previous affiliations of Scott M. Grundy include University of California, San Francisco & University of California, Davis.

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Effects of interruption of enterohepatic circulation on biliary lipid secretion in man.

TL;DR: The degree of coupling of cholesterol and phospholipids at low secretion of bile acids was variable from patient to patient and was a major determinant of the extent of increase in bile saturation.
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Comparisons of apolipoprotein B levels estimated by immunoassay, nuclear magnetic resonance, vertical auto profile, and non-high-density lipoprotein cholesterol in subjects with hypertriglyceridemia (SAFARI Trial).

TL;DR: More work is needed to improve agreement of apolipoprotein B measurements among methods employed clinically, and some improvement may be attained by taking into account the ratio of triglyceride/HDL cholesterol as a measurement of LDL particle size.
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Evaluation of a method for study of kinetics of autologous apolipoprotein A-I.

TL;DR: By isolation of autologous apoA-I under the conditions described, free apoC-I seemingly provides a valid method for estimating apoS-I turnover, and its use in turnover studies is described.
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Dissociation between postprandial lipemia and high density lipoprotein cholesterol concentrations in endurance-trained men.

TL;DR: The data indicate that the degree of postprandial lipemia is not the primary determinant of HDL cholesterol concentrations in endurance-trained men, and the wide range of LDL cholesterol concentrations measured in these men must be attributable to other factors.
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The -514 polymorphism in the hepatic lipase gene (LIPC) does not influence androgen-mediated stimulation of hepatic lipase activity

TL;DR: The data indicate that the -514 polymorphism does not influence the response of hepatic lipase activity to androgens, and that the effects of this polymorphism on hepaticlipase activity are independent of androgen action.