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Stanley Nattel
Researcher at Montreal Heart Institute
Publications - 802
Citations - 72437
Stanley Nattel is an academic researcher from Montreal Heart Institute. The author has contributed to research in topics: Atrial fibrillation & Heart failure. The author has an hindex of 132, co-authored 778 publications receiving 65700 citations. Previous affiliations of Stanley Nattel include Mayo Clinic & Brigham and Women's Hospital.
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Society Guidelines Canadian Cardiovascular Society Atrial Fibrillation Guidelines 2010: Rate and Rhythm Management
TL;DR: In this paper, the authors proposed the use of Beta-blockers or non-hydropyridine calcium channel blockers as the initial therapy for rate control of atrial fibrillation and atrial flutter in most patients without a history of myocardial infarction or left ventricular dysfunction.
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Insights into mechanisms linking cardiac hypertrophy and atrial fibrosis: evidence for a role of histone deacetylase in atrial fibrillation pathophysiology and therapy.
TL;DR: Strong evidence is presented demonstrating that, in addition to and distinct from the role of the RAAS, histone deacetylase (HDAC) overactivity causes atrial fibrosis, connexin-40 downregulation and atrial arrhythmia susceptibility in transgenic mice, and that pharmacologic inhibition of HDAC suppresses deleterious atrial remodeling.
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Pharmacotherapy for inherited arrhythmia syndromes: mechanistic basis, clinical trial evidence and practical application
Rafik Tadros,Julia Cadrin-Tourigny,Sylvia Abadir,Lena Rivard,Stanley Nattel,Mario Talajic,Paul Khairy +6 more
TL;DR: Current knowledge regarding drug therapy for inherited arrhythmias is reviewed and anti-arrhythmic mechanisms and available clinical evidence are highlighted while maintaining a practical perspective on patient management.
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Restoration of sinus rhythm in patients with atrial fibrillation
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Sudden Cardio Arrest: When normal ECG variants turn lethal
TL;DR: In 'Bedside to Bench', Stanley Nattel examines recent clinical studies suggesting that a particular type of readout on an electrocardiogram (ECG) may increase the risk of the condition.