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Stanley Nattel
Researcher at Montreal Heart Institute
Publications - 802
Citations - 72437
Stanley Nattel is an academic researcher from Montreal Heart Institute. The author has contributed to research in topics: Atrial fibrillation & Heart failure. The author has an hindex of 132, co-authored 778 publications receiving 65700 citations. Previous affiliations of Stanley Nattel include Mayo Clinic & Brigham and Women's Hospital.
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Journal ArticleDOI
Transmembrane ICa contributes to rate-dependent changes of action potentials in human ventricular myocytes.
TL;DR: Results suggest that reduction of Ca2+influx during the action potential accounts for most of the rate-dependent abbreviation of human ventricular APD.
Journal ArticleDOI
Differential distribution of Kir2.1 and Kir2.3 subunits in canine atrium and ventricle
TL;DR: Overall protein density differences are insufficient to explain I(K1) discrepancies, which may be related to differences in subcellular distribution.
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Redefining the Blanking Period After Catheter Ablation for Paroxysmal Atrial Fibrillation: Insights From the ADVICE (Adenosine Following Pulmonary Vein Isolation to Target Dormant Conduction Elimination) Trial
Stephan Willems,Paul Khairy,Jason G. Andrade,Boris A. Hoffmann,Sylvie Levesque,Atul Verma,Rukshen Weerasooriya,Paul Novak,Thomas Arentz,Isabel Deisenhofer,Thomas Rostock,Daniel Steven,Lena Rivard,Peter G. Guerra,Katia Dyrda,Blandine Mondésert,Marc Dubuc,Bernard Thibault,Mario Talajic,Denis Roy,Stanley Nattel,Laurent Macle +21 more
TL;DR: This study validates the use of a blanking period after catheter ablation for paroxysmal atrial fibrillation but calls into question the 90-day cut-off value.
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Differential Protein Kinase C Isoform Regulation and Increased Constitutive Activity of Acetylcholine-Regulated Potassium Channels in Atrial Remodeling
Samy Makary,Niels Voigt,Ange Maguy,Reza Wakili,Reza Wakili,Kunihiro Nishida,Masahide Harada,Dobromir Dobrev,Stanley Nattel,Stanley Nattel +9 more
TL;DR: These findings provide novel insights into mechanisms underlying IKAChC dysregulation in AF, with conventional Ca2+-dependent isoforms inhibiting and novel isoforms enhancing activity.
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Comparison of ion channel distribution and expression in cardiomyocytes of canine pulmonary veins versus left atrium.
TL;DR: Greater ERG and KvLQT1 abundance in pulmonary vein cardiomyocytes, lower abundance of Kir2.3 in pulmonary veins and differential pulmonary vein subcellular distribution of Kir 2.2 and Na(+),Ca(2+)-exchanger expression in PV are indicated.