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Stanley Nattel
Researcher at Montreal Heart Institute
Publications - 802
Citations - 72437
Stanley Nattel is an academic researcher from Montreal Heart Institute. The author has contributed to research in topics: Atrial fibrillation & Heart failure. The author has an hindex of 132, co-authored 778 publications receiving 65700 citations. Previous affiliations of Stanley Nattel include Mayo Clinic & Brigham and Women's Hospital.
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Journal ArticleDOI
Bevantolol disposition in patients with hepatic cirrhosis.
TL;DR: It is suggested that modifications in bevantolol dose should be considered when using this drug to treat patients with liver disease because of the high variability in its half‐life among cirrhotic patients.
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Sex as a Key Variable in Predicting Cardiovascular Outcomes: Rapidly Evolving Knowledge but Much More Needed.
Stanley Nattel,Louise Pilote +1 more
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Allele-Specific Gene Silencing: Another Step in the Inexorable Advance of Gene Therapy for Cardiac Arrhythmia Management.
TL;DR: A single injection of a cardiotropic adeno-associated virus carrying wild-type calsequestrin-2 is able to produce long-term suppression of the arrhythmic phenotype in mice with a loss-of-function mutation causing catecholaminergic polymorphic ventricular tachycardia (CPVT).
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The interaction between delayed rectifier channel alpha-subunits does not involve hetero-tetramer formation
Peter Biliczki,Andre Rüdiger,Zenawit Girmatsion,Marc Pourrier,Aida M. Mamarbachi,Terence E. Hébert,Ralf P. Brandes,Stefan H. Hohnloser,Stanley Nattel,Joachim R. Ehrlich +9 more
TL;DR: The results largely exclude the formation of hetero-tetramers between H2 and Q1 comprising the pore region or H2 C- or N-termini, and rather the transmembrane domains of the α-subunits appear relevant for the interaction.
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Modeling the aging heart: from local respiratory defects to global rhythm disturbances.
TL;DR: It is suggested that these cells provide "crystallization centers" for latent dysfunctional zones to allow arrhythmia emergence in widely distributed cells of the aging human heart.