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Ying Hu

Researcher at National Institutes of Health

Publications -  181
Citations -  17475

Ying Hu is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Cancer & Gene. The author has an hindex of 46, co-authored 156 publications receiving 14842 citations. Previous affiliations of Ying Hu include University of Maryland, Baltimore & Thomas Jefferson University.

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Comprehensive molecular portraits of human breast tumours

Daniel C. Koboldt, +355 more
- 04 Oct 2012 - 
TL;DR: The ability to integrate information across platforms provided key insights into previously defined gene expression subtypes and demonstrated the existence of four main breast cancer classes when combining data from five platforms, each of which shows significant molecular heterogeneity.
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Genetic Alterations Activating Kinase and Cytokine Receptor Signaling in High-Risk Acute Lymphoblastic Leukemia

Kathryn G. Roberts, +49 more
- 14 Aug 2012 - 
TL;DR: Several genetic alterations that activate kinase signaling in Ph-like ALL induce transformation that is attenuated with tyrosine kinase inhibitors, suggesting the treatment outcome of these patients may be improved with targeted therapy.
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Allelic Variation in Gene Expression Is Common in the Human Genome

H. Shuen Lo, +6 more
- 01 Aug 2003 - 
TL;DR: It is demonstrated that variation of gene expression between alleles is common, and this variation may contribute to human variability, as shown by real-time quantitative PCR experiments.
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Assessment of functional capacity before major non-cardiac surgery: an international, prospective cohort study

Duminda N. Wijeysundera, +180 more
- 30 Jun 2018 - 
TL;DR: Subjective assessment of functional capacity should not be used for preoperative risk evaluation, and Clinicians should instead consider a measure such as DASI for cardiac risk assessment.
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Susceptibility to hepatocellular carcinoma is associated with genetic variation in the enzymatic detoxification of aflatoxin B1

Katherine A McGlynn, +9 more
- 14 Mar 1995 - 
TL;DR: It is indicated that there is a synergistic increase in risk of HCC with the combination of hepatitis B virus infection and susceptible genotype and individuals with mutant genotypes at EPHX and GSTM1 may be at greater risk of developing AFB1 adducts, p53 mutations, and HCC when exposed to AFB1.