Appetite-regulatory hormone responses on the day following a prolonged bout of moderate-intensity exercise
James A. King,Jack O. Garnham,Andrew P. Jackson,Benjamin M. Kelly,Soteris Xenophontos,Myra A. Nimmo +5 more
TLDR
It is indicated that short-term energy deficits induced by exercise initially prompt a compensatory response by chronic but not acute hormonal regulators of appetite and energy balance within this 24h time-frame however there is no conscious recognition of the perturbation to energy balance.About:
This article is published in Physiology & Behavior.The article was published on 2015-03-15 and is currently open access. It has received 27 citations till now. The article focuses on the topics: Appetite.read more
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BRIEF COMMUNICATION ARISING: Gut hormone PYY3-36 physiologically inhibits food intake
Rachel L. Batterham,Michael A. Cowley,Caroline J. Small,Herbert Herzog,Mark A Cohen,C. L. Dakin,Alison M. Wren,Audrey E. Brynes,Malcolm J. Low +8 more
TL;DR: The authors showed that post-prandial elevation of PYY3-36 may act through the arcuate nucleus Y2R to inhibit feeding in a gut-hypothalamic pathway.
Journal ArticleDOI
Effects of exercise intensity on plasma concentrations of appetite-regulating hormones: Potential mechanisms
TL;DR: Blood redistribution during exercise may be important for suppressing ghrelin, while other mechanisms involving cytokine release, changes in plasma glucose and insulin concentrations, SNS activity, and muscle metabolism likely mediate changes in the anorexigenic signals PYY and GLP-1.
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Acute and Chronic Effects of Exercise on Appetite, Energy Intake, and Appetite-Related Hormones: The Modulating Effect of Adiposity, Sex, and Habitual Physical Activity.
James L. Dorling,David Broom,Stephen F. Burns,David J. Clayton,Kevin Deighton,Lewis J. James,James A. King,Masashi Miyashita,Alice E. Thackray,Rachel L. Batterham,David J. Stensel +10 more
TL;DR: The balance of evidence suggests that adiposity and sex do not modify appetite or energy intake responses to acute or chronic exercise interventions, but individuals with higher habitual physical activity levels may better adjust energy intake in response to energy balance perturbations.
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The Unexplored Crossroads of the Female Athlete Triad and Iron Deficiency: A Narrative Review
TL;DR: How iron deficiency may interact with each component of the female athlete triad, that is, energy status, reproductive function, and bone health, is described.
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Weight Management for Athletes and Active Individuals: A Brief Review
TL;DR: The concept of dynamic energy balance is reviewed, including two mathematical models developed to improve weight-loss predictions based on changes in diet and exercise and these models are now available on the Internet.
References
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Responses of Leptin to Short-Term Fasting and Refeeding in Humans: A Link With Ketogenesis but Not Ketones Themselves
Jerzy W. Kolaczynski,Robert V. Considine,Joanna P. Ohannesian,Cheryl C. Marco,Irina Opentanova,Mark R. Nyce,Michael Myint,Jasé F Caro +7 more
TL;DR: The study indicates that one of the adaptive physiological responses to fasting is a fall in serum leptin, although the mediator that brings about this effect remains unknown, it appears to be neither insulin nor ketones.
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Relationship Between Insulin Sensitivity and Plasma Leptin Concentration in Lean and Obese Men
TL;DR: It is demonstrated that insulin resistance is associated with elevated plasma leptin levels independent of body fat mass, however, plasma insulin itself does not acutely regulate leptin production.
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Plasma ghrelin levels and hunger scores in humans initiating meals voluntarily without time- and food-related cues.
TL;DR: The preprandial increase of ghrelin levels that the authors observed among humans initiating meals voluntarily, without time- or food-related cues, and the overlap between these levels and hunger scores are consistent with a role for gh Relin in meal initiation.
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Hexosamines, insulin resistance, and the complications of diabetes: current status
TL;DR: Increased flux through HBP is required and sufficient for some of the metabolic effects of sustained, increased glucose flux, which promotes the complications of diabetes, e.g., diminished expression of sarcoplasmic reticulum Ca(2+)-ATPase in cardiomyocytes and induction of TGF-beta and plasminogen activator inhibitor-1 in vascular smooth muscle cells, mesangial cells, and aortic endothelial cells.
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Exercise training-induced improvements in insulin action
John A. Hawley,Sarah J. Lessard +1 more
TL;DR: Evidence now suggests that the improvements in insulin sensitivity associated with exercise training are also related to changes in the expression and/or activity of proteins involved in insulin signal transduction in skeletal muscle such as the AMP‐activated protein kinase (AMPK) and theprotein kinase B (Akt) substrate AS160.