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Infectious Diseases of Humans: Dynamics and Control

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TLDR
This book discusses the biology of host-microparasite associations, dynamics of acquired immunity heterogeneity within the human community indirectly transmitted helminths, and the ecology and genetics of hosts and parasites.
Abstract
Part 1 Microparasites: biology of host-microparasite associations the basic model - statics static aspects of eradication and control the basic model - dynamics dynamic aspects of eradication and control beyond the basic model - empirical evidence of inhomogeneous mixing age-related transmission rates genetic heterogeneity social heterogeneity and sexually transmitted diseases spatial and other kinds of heterogeneity endemic infections in developing countries indirectly transmitted microparasites. Part 2 Macroparasites: biology of host-macroparasite associations the basic model - statics the basic model - dynamics acquired immunity heterogeneity within the human community indirectly transmitted helminths experimental epidemiology parasites, genetic variability, and drug resistance the ecology and genetics of host-parasite associations.

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Journal ArticleDOI

Contrasting molecular epidemiology of group A streptococci causing tropical and nontropical infections of the skin and throat.

TL;DR: Analysis of data supports the hypothesis that GAS tissue preferences are genetically predetermined and that host risk factors for infection strongly influence the differential reproduction of individual clones.
Journal Article

Integrating association data and disease dynamics in a social ungulate: Bovine tuberculosis in African buffalo in the Kruger National Park

TL;DR: In this paper, the authors explored how non-random association patterns (i.e. who spends time with whom) affect disease dynamics in African buffalo herds. But, the authors did not consider the association data in a network disease model in a wildlife population.
Journal ArticleDOI

2009 H1N1 influenza.

TL;DR: The 2009 H1N1 pandemic caused the first influenza pandemic in decades as mentioned in this paper, which has caused disproportionate disease among young people with early reports of virulence similar to that of seasonal influenza.
Journal ArticleDOI

Predicting the local dynamics of epizootic rabies among raccoons in the United States

TL;DR: The best quantitative agreement between data and model assumed low levels of immunity within raccoon populations, suggesting that raccoons develop little or no rabies immune class.
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