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Infectious Diseases of Humans: Dynamics and Control
Roy M. Anderson,Robert M. May +1 more
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TLDR
This book discusses the biology of host-microparasite associations, dynamics of acquired immunity heterogeneity within the human community indirectly transmitted helminths, and the ecology and genetics of hosts and parasites.Abstract:
Part 1 Microparasites: biology of host-microparasite associations the basic model - statics static aspects of eradication and control the basic model - dynamics dynamic aspects of eradication and control beyond the basic model - empirical evidence of inhomogeneous mixing age-related transmission rates genetic heterogeneity social heterogeneity and sexually transmitted diseases spatial and other kinds of heterogeneity endemic infections in developing countries indirectly transmitted microparasites. Part 2 Macroparasites: biology of host-macroparasite associations the basic model - statics the basic model - dynamics acquired immunity heterogeneity within the human community indirectly transmitted helminths experimental epidemiology parasites, genetic variability, and drug resistance the ecology and genetics of host-parasite associations.read more
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Automated monitoring of behavior reveals bursty interaction patterns and rapid spreading dynamics in honeybee social networks.
Tim Gernat,Vikyath D. Rao,Martin Middendorf,Harry Dankowicz,Nigel Goldenfeld,Gene E. Robinson +5 more
TL;DR: It is found that bees, like humans, also interact in bursts but that spreading is significantly faster than in a randomized reference network and remains so even after an experimental demographic perturbation, indicating that burstiness may be an intrinsic property of social interactions, but it does not always inhibit spreading in real-world communication networks.
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The noisy voter model on complex networks
TL;DR: It is shown how the degree heterogeneity—variance of the underlying degree distribution—has a strong influence on the location of the critical point of a noise-induced, finite-size transition occurring in the model, on the local ordering of the system, and on the functional form of its temporal correlations.
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HIV-1 dynamics revisited: biphasic decay by cytotoxic T lymphocyte killing?
TL;DR: It is shown that the biphasic decay can be explained simply, without invoking multiple compartments: viral load falls quickly while cytotoxic T lymphocytes (CTL) are still abundant, and more slowly as CTL disappear.
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Seventy-five years of estimating the force of infection from current status data
TL;DR: An historical overview is presented, discussing the relevance of Muench's work, and the wide array of newer methods with illustrations on pre-vaccination serological survey data of two airborne infections: rubella and parvovirus B19 are explained.
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Modelling measles re-emergence as a result of waning of immunity in vaccinated populations
Joël Mossong,Claude P. Muller +1 more
TL;DR: Using current estimates of duration of vaccine-derived protection, measles would not be expected to re-emerge quickly in countries with sustained high routine vaccine coverage, however, re-emergence is possible to occur several decades after introduction of high levels of vaccination.