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Claude Bouchard

Researcher at Pennington Biomedical Research Center

Publications -  1105
Citations -  121841

Claude Bouchard is an academic researcher from Pennington Biomedical Research Center. The author has contributed to research in topics: Body mass index & Obesity. The author has an hindex of 153, co-authored 1076 publications receiving 115307 citations. Previous affiliations of Claude Bouchard include Texas A&M University & University of Texas at Austin.

Papers
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Two ethnic-specific polymorphisms in the human Agouti-related protein gene are associated with macronutrient intake

TL;DR: The present study suggests that 2 ethnic-specific AGRP variants, previously shown to be associated with leanness in the HERITAGE Family Study, are also associated with macronutrient intake.
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Race differences in the response of postheparin plasma lipoprotein lipase and hepatic lipase activities to endurance exercise training in men: results from the HERITAGE Family Study.

TL;DR: Results of the present study suggest that in Whites, the increase in PH-LPL activity in response to endurance exercise training is associated with a better lipoprotein-lipid profile, therefore reducing CHD risk, however, the generally better metabolic profile of Black individuals may minimize further improvement of lipoproteins by exercise training.
Book

Regulation of body weight : biological and behavioral mechanisms : report of the Dahlem Workshop on Regulation of Body Weight: Biological and Behavioral Mechanisms, Berlin, May 14-19, 1995

TL;DR: What are the Animal and Human Models for the Study of Regulation of Body Weight and What Are Their Respective Strenghts and Limitations?
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The T111I mutation in the EL gene modulates the impact of dietary fat on the HDL profile in women.

TL;DR: The gene-diet interaction among women suggests that the T111I missense mutation may confer protection against the lowering effect of a high dietary PUFA intake on plasma apoA-I and HDL3-C levels.
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Submaximal power output in adopted and biological siblings.

TL;DR: It is concluded that submaximal power output is only moderately affected by the genotypes of free-living children, adolescents and young adults.