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Edythe D. London

Researcher at University of California, Los Angeles

Publications -  492
Citations -  36481

Edythe D. London is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Nicotinic agonist & Methamphetamine. The author has an hindex of 93, co-authored 482 publications receiving 33741 citations. Previous affiliations of Edythe D. London include Icahn School of Medicine at Mount Sinai & Semel Institute for Neuroscience and Human Behavior.

Papers
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Journal ArticleDOI

Brain nicotinic acetylcholine receptor occupancy: effect of smoking a denicotinized cigarette.

TL;DR: Analysis of PET data revealed that nicotine inhalation during smoking appears to be solely responsible for alpha4beta2* nAChR occupancy, with other factors (if present at all) having either short-lived or very minor effects.
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Neural Correlates of Affect Processing and Aggression in Methamphetamine Dependence

TL;DR: Contrary to the hypotheses, methamphetamine-dependent individuals may successfully regulate emotions through incidental means (affect labeling) and low ventral inferior frontal gyrus activity may contribute to heightened aggression by limiting emotional insight.
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Methamphetamine dependence and neuropsychological functioning: evaluating change during early abstinence.

TL;DR: The findings suggest that methamphetamine-dependent subjects do not show considerable cognitive gains in the first month of abstinence, and a greater length of abstinence may be needed for cognitive improvement.
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Methamphetamine craving induced in an online virtual reality environment.

TL;DR: A physiological divergence between high and low craving METH abusers using heart rate variability is revealed, and the usefulness of VR cues for eliciting subjective craving in Meth abusers is demonstrated, as well as the effectiveness of a novel VR drug cue model created within an online virtual world.
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Neural Correlates of Response Inhibition and Cigarette Smoking in Late Adolescence

TL;DR: Findings suggest that smoking can modulate prefrontal cortical function, and it is possible that smoking may influence the trajectory of brain development during this critical developmental period.