E
Edythe D. London
Researcher at University of California, Los Angeles
Publications - 492
Citations - 36481
Edythe D. London is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Nicotinic agonist & Methamphetamine. The author has an hindex of 93, co-authored 482 publications receiving 33741 citations. Previous affiliations of Edythe D. London include Icahn School of Medicine at Mount Sinai & Semel Institute for Neuroscience and Human Behavior.
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Effects of Leptin Deficiency and Replacement on Cerebellar Response to Food-Related Cues
TL;DR: It is indicated that leptin replacement reversibly alters neural function within the posterior cerebellum and modulates plasticity-dependent brain physiology in response to food cues, suggesting an underexplored role for the posterior Cerebellum in the regulation of leptin-mediated processes related to food intake.
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Intravenous Cocaine Decreases Cardiac Vagal Tone, Vagal Index (Derived in Lorenz Space), and Heart Period Complexity (Approximate Entropy) in Cocaine Abusers
TL;DR: Findings, and evidence that cocaine decreased cardiovascular complexity, contradict the prevailing assumption that the mechanism by which cocaine produces tachycardia is sympathetic (beta-adrenergic) and discuss implications for cardiac arrhythmias associated with cocaine abuse and death due to overdose.
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Functional Genetic Variation in Dopamine Signaling Moderates Prefrontal Cortical Activity During Risky Decision-Making
Milky Kohno,Erika L. Nurmi,Christopher P Laughlin,Angelica M. Morales,Emma H Gail,Gerhard Hellemann,Edythe D. London +6 more
TL;DR: The findings support the view that the links between dopaminergic signaling, prefrontal function, and decision making vary as a function of dopamine signaling capacity.
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Self-administration of 5-iodo-A-85380, a β2-selective nicotinic receptor ligand, by operantly trained rats
TL;DR: This finding suggests that interaction with &bgr;2-containing nAChRs, without direct involvement of &agr;7 receptors, can produce reinforcement and thereby can support self-administration behavior.
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Differential effects of μ and κ opioid analgesics on cerebral glucose utilization in the rat
TL;DR: The findings suggest that different supraspinal mechanisms are involved in the actions of μ vs κ opioids, and indicate that the [14C]2-DG procedure might be helpful in elucidating the anatomical areas involved.