E
Edythe D. London
Researcher at University of California, Los Angeles
Publications - 492
Citations - 36481
Edythe D. London is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Nicotinic agonist & Methamphetamine. The author has an hindex of 93, co-authored 482 publications receiving 33741 citations. Previous affiliations of Edythe D. London include Icahn School of Medicine at Mount Sinai & Semel Institute for Neuroscience and Human Behavior.
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Journal ArticleDOI
Dopamine D2/D3 receptors play a specific role in the reversal of a learned visual discrimination in monkeys
TL;DR: The results strongly suggest that D2/D3 receptors, but not D1/D5 receptors, selectively mediate reversal learning, without affecting the capacity to learn a new stimulus-reward association.
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Working memory in cigarette smokers: comparison to non-smokers and effects of abstinence.
Adrianna Mendrek,John Monterosso,Sara L. Simon,Murray E. Jarvik,Arthur L. Brody,Richard G. Olmstead,Catherine P. Domier,Mark S. Cohen,Monique Ernst,Edythe D. London +9 more
TL;DR: Results indicated that performance of smokers after >or=13 h but not
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Neuropsychological functioning in opiate-dependent subjects receiving and following methadone maintenance treatment.
James Prosser,Lisa J. Cohen,Matthew Steinfeld,Daniel P. Eisenberg,Edythe D. London,Igor Galynker +5 more
TL;DR: The hypothesis that former heroin users who have detoxified from methadone maintenance therapy and are drug-free have less pronounced cognitive impairment than patients continuing long-term MMT is tested.
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Chronic methamphetamine abuse and corticostriatal deficits revealed by neuroimaging
TL;DR: These studies indicate that individuals with a history of chronic methamphetamine abuse often display several signs of corticostriatal dysfunction, including abnormal gray- and white-matter integrity, monoamine neurotransmitter system deficiencies, neuroinflammation, poor neuronal integrity, and aberrant patterns of brain connectivity and function, both when engaged in cognitive tasks and at rest.
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Supraphysiological doses of levothyroxine alter regional cerebral metabolism and improve mood in bipolar depression.
Michael Bauer,Michael Bauer,Edythe D. London,Natalie L. Rasgon,Natalie L. Rasgon,Steven M. Berman,Mark A. Frye,Lori L. Altshuler,M. Mandelkern,Jennifer Bramen,Bradley Voytek,Roger P. Woods,John C. Mazziotta,Peter C. Whybrow +13 more
TL;DR: B bipolar depressed patients have abnormal function in prefrontal and limbic brain areas and L-T4 may improve mood by affecting circuits involving these areas, which have been previously implicated in affective disorders.