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Esther Meissner
Researcher at Max Planck Society
Publications - 4
Citations - 737
Esther Meissner is an academic researcher from Max Planck Society. The author has contributed to research in topics: Dementia & Genome-wide association study. The author has an hindex of 4, co-authored 4 publications receiving 672 citations.
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Journal ArticleDOI
Comprehensive research synopsis and systematic meta-analyses in Parkinson's disease genetics: the PDGene database.
Christina M. Lill,Johannes T. Roehr,Johannes T. Roehr,Matthew B. McQueen,Fotini K. Kavvoura,Fotini K. Kavvoura,Fotini K. Kavvoura,Sachin Bagade,Brit-Maren M. Schjeide,Leif M. Schjeide,Esther Meissner,Ute Zauft,Nicole C. Allen,Tian-Jing Liu,Marcel Schilling,Marcel Schilling,Kari J. Anderson,Gary W. Beecham,Daniela Berg,Daniela Berg,Joanna M. Biernacka,Alexis Brice,Anita L. DeStefano,Chuong B. Do,Nicholas Eriksson,Stewart A. Factor,Matthew J. Farrer,Tatiana Foroud,Thomas Gasser,Thomas Gasser,Taye H. Hamza,John Hardy,Peter Heutink,Erin M. Hill-Burns,Christine Klein,Jeanne C. Latourelle,Demetrius M. Maraganore,Eden R. Martin,Maria Martinez,Maria Martinez,Richard H. Myers,Mike A. Nalls,Nathan Pankratz,Haydeh Payami,Wataru Satake,William K. Scott,Manu Sharma,Manu Sharma,Andrew B. Singleton,Kari Stefansson,Tatsushi Toda,Joyce Y. Tung,Jeffery M. Vance,Nicholas W. Wood,Cyrus P. Zabetian,Peter Young,Rudolph E. Tanzi,Muin J. Khoury,Frauke Zipp,Hans Lehrach,John P. A. Ioannidis,Lars Bertram,Lars Bertram +62 more
TL;DR: This study provides an exhaustive and up-to-date summary of the status of PD genetics research that can be readily scaled to include the results of future large-scale genetics projects, including next-generation sequencing studies.
Journal ArticleDOI
The role of TREM2 R47H as a risk factor for Alzheimer's disease, frontotemporal lobar degeneration, amyotrophic lateral sclerosis, and Parkinson's disease.
Christina M. Lill,Christina M. Lill,Aina Rengmark,Lasse Pihlstrøm,Isabella Fogh,Aleksey Shatunov,Patrick M. A. Sleiman,Patrick M. A. Sleiman,Li-San Wang,Tian Liu,Christina F. Lassen,Esther Meissner,Panos Alexopoulos,Andrea Calvo,Adriano Chiò,Nil Dizdar,Frank Faltraco,Lars Forsgren,Julia Kirchheiner,Alexander Kurz,Jan Petter Larsen,Maria Liebsch,Jan Linder,Karen E. Morrison,Karen E. Morrison,Hans Nissbrandt,Markus Otto,Jens Pahnke,Jens Pahnke,Amanda Partch,Gabriella Restagno,Dan Rujescu,Cathrin Schnack,Christopher Shaw,Pamela J. Shaw,Hayrettin Tumani,Ole-Bjørn Tysnes,Ole-Bjørn Tysnes,Otto Valladares,Vincenzo Silani,Leonard H. van den Berg,Wouter van Rheenen,Jan H. Veldink,Ulman Lindenberger,Elisabeth Steinhagen-Thiessen,Stefan J. Teipel,Stefan J. Teipel,Robert Perneczky,Robert Perneczky,Hakon Hakonarson,Hakon Hakonarson,Harald Hampel,Harald Hampel,Christine A. F. von Arnim,Jørgen H. Olsen,Vivianna M. Van Deerlin,Ammar Al-Chalabi,Mathias Toft,Beate Ritz,Lars Bertram,Lars Bertram,Lars Bertram +61 more
TL;DR: The data show that rs75932628 is highly significantly associated with the risk of AD across 24,086 AD cases and 148,993 controls of European descent and carriers of the rs75 932628 risk allele showed significantly increased levels of CSF‐total‐tau but not Aβ42 suggesting that TREM2's role in AD may involve tau dysfunction.
Journal ArticleDOI
Closing the case of APOE in multiple sclerosis: no association with disease risk in over 29 000 subjects
Christina M. Lill,Tian Liu,Brit-Maren M. Schjeide,Johannes T. Roehr,Denis A. Akkad,Vincent Damotte,Antonio Alcina,Miguel A. Ortiz,R. Arroyo,Aitzkoa Lopez de Lapuente,Paul Blaschke,Alexander Winkelmann,Lisa-Ann Gerdes,Felix Luessi,Oscar Fernadez,Guillermo Izquierdo,Alfredo Antigüedad,Sabine Hoffjan,Isabelle Cournu-Rebeix,Silvana Gromöller,H. Faber,Maria Liebsch,Esther Meissner,Coralie Chanvillard,Emmanuel Touzé,Thomas Dörner,R Heekeren,Ulman Lindenberger,Andrew T. Chan,Peter Lohse,Christian Kubisch,Uwe K. Zettl,Elena Urcelay,Lars Bertram,Frauke Zipp +34 more
TL;DR: It is unlikely that the two APOE missense SNPs studied here exert any relevant effects on MS susceptibility, given the large sample size of the analyses.
Journal ArticleDOI
Alzheimer's disease pathology explains association between dementia with Lewy bodies and APOE-ε4/TOMM40 long poly-T repeat allele variants.
Inga Prokopenko,Inga Prokopenko,Gentaro Miyakawa,Gentaro Miyakawa,Bang Zheng,Jani Heikkinen,Daniela Petrova Quayle,Chinedu T. Udeh-Momoh,Annique Claringbould,Juliane Neumann,Hazal Haytural,Marika Kaakinen,Marika Kaakinen,Elena Loizidou,Esther Meissner,Lars Bertram,Lars Bertram,Djordje Gveric,Steve M. Gentleman,Johannes Attems,Robert Perneczky,Robert Perneczky,Robert Perneczky,Thomas Arzberger,Pierandrea Muglia,Christina M. Lill,Christina M. Lill,Laura Parkkinen,Lefkos T. Middleton +28 more
TL;DR: The role of TOMM40‐APOE 19q13.3 region variants in Alzheimer's disease and dementia is well documented but remains contentious in dementia with Lewy bodies and Parkinson's disease dementia.