E
Eugene Braunwald
Researcher at Brigham and Women's Hospital
Publications - 1758
Citations - 278949
Eugene Braunwald is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Myocardial infarction & TIMI. The author has an hindex of 230, co-authored 1711 publications receiving 264576 citations. Previous affiliations of Eugene Braunwald include Boston University & University of California, San Francisco.
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Suppression of Tumorigenicity 2 in Heart Failure With Preserved Ejection Fraction.
Omar F. AbouEzzeddine,Paul M. McKie,Shannon M. Dunlay,Susanna R. Stevens,G. Michael Felker,Barry A. Borlaug,Horng H. Chen,Russell P. Tracy,Eugene Braunwald,Margaret M. Redfield +9 more
TL;DR: In HFpEF, sST2 levels were associated with proinflammatory comorbidities, right ventricular pressure overload and dysfunction, and systemic congestion but not with left ventricular geometry or function, suggesting that ST2 may be a marker of systemic inflammation inHFpEF and potentially of extracardiac origin.
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Clinical Benefit of Cardiorenal Effects of Sodium-Glucose Cotransporter 2 Inhibitors: JACC State-of-the-Art Review
TL;DR: This second part of this 2-part review summarizes the findings of recent clinical trials and their clinical implications and describes ongoing trials and future areas of research.
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Studies on Starling's law of the heart. V. Left ventricular function in man.
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Prospective Evaluation of Pregnancy-Associated Plasma Protein-A and Outcomes in Patients With Acute Coronary Syndromes
Marc P. Bonaca,Benjamin M. Scirica,Marc S. Sabatine,Petr Jarolim,Sabina A. Murphy,Janna S. Chamberlin,Daniel W. Rhodes,Paula C. Southwick,Eugene Braunwald,David A. Morrow +9 more
TL;DR: PAPP-A was independently associated with recurrent cardiovascular events in patients with NSTE-ACS and supports PAPP- A as a candidate prognostic marker in Patients with ACS and supports investigation of its therapeutic implications.
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Sustained, paired electrical stimuli: slowing of the ventricular rate and augmentation of contractile force.
TL;DR: Paired electrical stimuli to the ventricles result in profound alterations in both the electrical and contractile properties of the mammalian heart, and it is possible that this technic will be applicable and beneficial in certain patients, when a slower heart rate and/or augmented myocardial contractile force is desirable.