J
Jason G. Cyster
Researcher at University of California, San Francisco
Publications - 214
Citations - 46949
Jason G. Cyster is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: B cell & Germinal center. The author has an hindex of 106, co-authored 199 publications receiving 42370 citations. Previous affiliations of Jason G. Cyster include London Research Institute & Howard Hughes Medical Institute.
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Epstein-Barr Virus–induced Molecule 1 Ligand Chemokine Is Expressed by Dendritic Cells in Lymphoid Tissues and Strongly Attracts Naive T Cells and Activated B Cells
TL;DR: A central role for ELC is suggested in promoting encounters between recirculating T cells and dendritic cells and in the migration of activated B cells into the T zone of secondary lymphoid tissues.
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FTY720: sphingosine 1-phosphate receptor-1 in the control of lymphocyte egress and endothelial barrier function.
TL;DR: The novel immunomodulator FTY720 acts as a high‐affinity agonist at the G protein‐coupled sphingosine 1‐phosphate receptor‐1 on thymocytes and lymphocytes, thereby inducing aberrant internalization of the receptor.
Journal ArticleDOI
Follicular stromal cells and lymphocyte homing to follicles.
Jason G. Cyster,K. M. Ansel,Karin Reif,Eric H. Ekland,P. L. Hyman,H. L. Tang,Sanjiv A. Luther,Vu N. Ngo +7 more
TL;DR: How antigen recognition causes T and B lymphocytes to undergo changes in chemokine responsiveness that may help direct their movements into, or out of, lymphoid follicles is considered.
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BLC expression in pancreatic islets causes B cell recruitment and lymphotoxin-dependent lymphoid neogenesis.
TL;DR: It is established that BLC is sufficient to activate a pathway of events leading to formation of organized lymphoid tissue.
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B Cell Responses: Cell Interaction Dynamics and Decisions
TL;DR: How basic research is informing efforts to generate vaccines that induce broadly neutralizing antibodies against viral pathogens is discussed, revealing the special features associated with allergen-reactive IgE responses and uncovering the antibody-independent mechanisms by which B cells contribute to health and disease.