M
Mark Hallett
Researcher at National Institutes of Health
Publications - 1234
Citations - 136876
Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.
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Cortical mechanisms of recovery of function after stroke
TL;DR: Techniques, such as transcranial magnetic stimulation and neuroimaging with positron emission tomography, can be used to demonstrate the organization of the human motor system and do reveal plastic reorganization in a number of situations.
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Neural underpinning of postural responses to visual field motion.
TL;DR: The existence of functional interactions between modality specific areas of the brain involved in postural responses to Visual Field Motion (VFM) is proposed.
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Differentiation of sensorimotor neuronal structures responsible for induction of motor evoked potentials, attenuation in detection of somatosensory stimuli, and induction of sensation of movement by mapping of optimal current directions
TL;DR: It is concluded that neuronal networks targeting corticospinal neurons responsible for MEPs are different from those leading to SOM and ADSS (which could not be differentiated), which were not differentiated.
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Metabolic alterations in corpus callosum may compromise brain functional connectivity in MTBI patients: an 1H-MRS study.
Brian F. G. Johnson,Kai Zhang,Thomas Neuberger,Silvina G. Horovitz,Mark Hallett,Wayne J. Sebastianelli,Semyon Slobounov,Semyon Slobounov +7 more
TL;DR: This novel finding supports the hypothesis that the functional disruption of interhemispheric brain networks in MTBI subjects results from compromised metabolic integrity of the corpus callosum and that this persists despite apparent clinical return to baseline.
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Cerebellar diaschisis revisited: pontine hypometabolism and dentate sparing.
TL;DR: Analyzing glucose metabolism in the posterior fossa in 67 patients with primary unilateral supratentorial brain tumors found that glucose utilization was lower in the contralateral cerebellar cortex (compared with the ipsilateral one), consistent with the selection criterion, and the “dentate sparing” suggests preservation of afferent input to the largest of the deep Cerebellar nuclei from the Purkinje cells in the cortex.