M
Mark Hallett
Researcher at National Institutes of Health
Publications - 1234
Citations - 136876
Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.
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Journal ArticleDOI
Movement-related electroencephalographic desynchronization in patients with hand cramps: evidence for motor cortical involvement in focal dystonia.
TL;DR: Because EEG beta rhythm in the sensorimotor region likely emanates from the motor cortex and is related to ongoing muscle activity, this abnormality could be a manifestation of the abnormal motor command at the cortical level.
Book ChapterDOI
Explanation as treatment for functional neurologic disorders
TL;DR: There are a number of generic components to effective explanation shared by most authors on the topic that form the basis of a consensus, including taking the patient seriously, giving the problem a diagnostic label, explaining the rationale for the diagnosis, and effective triage and referral for other treatment where appropriate.
Journal ArticleDOI
Tricks in dystonia: ordering the complexity
TL;DR: It is postulated that sensory tricks decrease abnormally increased facilitation to inhibition ratios in the dystonic brain, and it appears worthwhile for patients to search for possible sensory tricks.
Journal ArticleDOI
Role of intracortical mechanisms in the late part of the silent period to transcranial stimulation of the human motor cortex
TL;DR: Findings support the idea that a significant component of the SP arises from intracortical mechanisms, as TMS is more effective than TES or PBS in activating cortical interneurons.
Journal ArticleDOI
Movement‐related cortical potentials in patients with cerebellar degeneration
TL;DR: The patterns found in this preliminary study indicate a derangement of sensorimotor cortex activity in voluntary movement as a consequence of cerebellar dysfunction.