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Mark Hallett

Researcher at National Institutes of Health

Publications -  1234
Citations -  136876

Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.

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A finite element analysis of the effect of electrode area and inter-electrode distance on the spatial distribution of the current density in tDCS

TL;DR: It is found that smaller electrodes required slightly less current to achieve a constant value of the current density at a reference point on the brain surface located directly under the electrode center, and electrodes with areas in the range 3.5-12 cm(2) may provide a better compromise between focality and current density in the scalp than the traditional electrodes.
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Volitional Control of Movement: The Physiology of Free Will

TL;DR: The evidence suggests that movement is initiated in the frontal lobe, particularly the mesial areas, and the sense of volition arises as the result of a corollary discharge likely involving multiple areas with reciprocal connections including those in the parietal lobe and insular cortex.
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A PET study of sequential finger movements of varying length in patients with Parkinson's disease.

TL;DR: It is concluded that in Parkinson's disease patients more cortical areas are recruited to perform sequential finger movements; this may be the result of increasing corticocortical activity to compensate for striatal dysfunction.
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Dopamine agonists and risk: impulse control disorders in Parkinson's; disease

TL;DR: Dopamine agonists enhance sensitivity to risk in patients with impulse control disorder possibly by impairing risk evaluation in the striatum, providing a potential explanation of why dopamine agonists may lead to an unconscious bias towards risk in susceptible individuals.
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Deficient activation of the motor cortical network in patients with writer’s cramp

TL;DR: Deficient activation of premotor cortex and decreased correlation between prem motor cortical regions and putamen suggest a dysfunction of the premotor cortical network in patients with writer’s cramp possibly arising in the basal ganglia, compatible with a loss of inhibition during the generation of motor commands.