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Mark Hallett

Researcher at National Institutes of Health

Publications -  1234
Citations -  136876

Mark Hallett is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transcranial magnetic stimulation & Motor cortex. The author has an hindex of 186, co-authored 1170 publications receiving 123741 citations. Previous affiliations of Mark Hallett include Government of the United States of America & Armed Forces Institute of Pathology.

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Leg paresthesias induced by magnetic brain stimulation in patients with thoracic spinal cord injury

TL;DR: It is suggested that portions of the cortical representation areas for body parts deafferented by a complete spinal cord injury can remain related to those body parts for up to several years.
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Cortical areas with enhanced activation during object-centred spatial information processing. A PET study.

TL;DR: The involvement of the occipitotemporal cortex and a broad frontoparietal network when, as in the visuomotor task, object-centred information guides movement is suggested, when the same data underlie declarative reports.
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Parametric analysis of functional neuroimages: application to a variable-rate motor task.

TL;DR: A method for mathematically modeling the changes in regional cerebral blood flow (rCBF) as a function of experimental parameters using step and linear functions is presented.
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Cortical activation during fast repetitive finger movements in humans: dipole sources of steady-state movement-related cortical potentials.

TL;DR: Steady-state movement-related cortical potentials in combination with dipole modelling provide a novel, noninvasive approach to assessing changes of human cortical premotor, motor, and somatosensory activation in the millisecond range.
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Suppression of vision by transcranial magnetic stimulation: a third mechanism.

TL;DR: Three periods when single-pulse transcranial magnetic stimulation of the occipital pole impaired performance on a forced-choice visual letter-identification task are reported, with the most likely explanation being a blink-associated interference with letter-processing neural activity.