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Michael F. Gurish

Researcher at Brigham and Women's Hospital

Publications -  105
Citations -  10168

Michael F. Gurish is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Mast cell & Proteases. The author has an hindex of 56, co-authored 105 publications receiving 9185 citations. Previous affiliations of Michael F. Gurish include Harvard University & Boston Children's Hospital.

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Mast Cells: A Cellular Link Between Autoantibodies and Inflammatory Arthritis

TL;DR: Two strains of mice deficient in mast cells were resistant to development of joint inflammation and that susceptibility was restored in the W/Wv strain by mast cell engraftment, suggesting mast cells may function as a cellular link between autoantibodies, soluble mediators, and other effector populations in inflammatory arthritis.
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Heparin is essential for the storage of specific granule proteases in mast cells

TL;DR: Transgenic mice generated by disrupting the N -deacetylase/N -sulphotransferase-2 gene are described, that cannot express fully sulphated heparin and are shown to controls, through a post-translational mechanism, the levels of specific cassettes of positively charged proteases inside mast cells.
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Developmental checkpoints of the basophil/mast cell lineages in adult murine hematopoiesis

TL;DR: In this article, the β7 integrin was found to be required for selective homing of mast cell progenitors to the periphery of the mouse spleen, and the granulocyte-related transcription factor CCAAT/enhancer-binding protein α (C/EBPα) played a primary role in the fate decision of BMCPs, being expressed in BaPs but not in MCPs.
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Cooperative and antagonistic interplay between PU.1 and GATA-2 in the specification of myeloid cell fates.

TL;DR: A developmental model in which cooperative function or antagonistic crossregulation by PU.1 and GATA transcription factors appear to antagonize each other's function in the development of distinct lineages of the hematopoietic system is proposed.