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Thomas Foltynie

Researcher at UCL Institute of Neurology

Publications -  381
Citations -  26959

Thomas Foltynie is an academic researcher from UCL Institute of Neurology. The author has contributed to research in topics: Deep brain stimulation & Parkinson's disease. The author has an hindex of 80, co-authored 352 publications receiving 21023 citations. Previous affiliations of Thomas Foltynie include University College London & University of Kent.

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Subthalamic nucleus gamma activity increases not only during movement but also during movement inhibition

TL;DR: Investigating the role of gamma during fast stopping and recorded scalp electroencephalogram and local field potentials from deep brain stimulation electrodes in 9 Parkinson's disease patients found that STN gamma activity may support flexible motor control as it did not only increase during movement execution but also during rapid action-stopping.
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Post hoc analysis of the Exenatide-PD trial-Factors that predict response.

TL;DR: While patients with a range of demographic and clinical factors can potentially benefit from exenatide once‐weekly, data support an emphasis towards recruiting patients at earlier disease in future planned clinical trials of gluacagon‐like peptide‐1 (GLP‐1) receptor agonists in Parkinson's disease.
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Proceedings of the Seventh Annual Deep Brain Stimulation Think Tank: Advances in Neurophysiology, Adaptive DBS, Virtual Reality, Neuroethics and Technology

TL;DR: The Seventh Annual Deep Brain Stimulation Think Tank addressed the most current use and utility of complex neurophysiological signals for development of adaptive neurostimulation to improve clinical outcomes and ethical issues arising in and from research and use of DBS.
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Impact of GBA1 variants on long-term clinical progression and mortality in incident Parkinson’s disease

TL;DR: GBA1 variants, including those not associated with Gaucher disease, are common in PD and result in a more aggressive disease course and pathogenic and ‘non-pathogenic’ GBA1 variant were associated with the accelerated development of dementia and a moreaggressive motor course.