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William W. Busse
Researcher at University of Wisconsin-Madison
Publications - 740
Citations - 62685
William W. Busse is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Asthma & Eosinophil. The author has an hindex of 115, co-authored 697 publications receiving 56703 citations. Previous affiliations of William W. Busse include National Institutes of Health & University at Buffalo.
Papers
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Journal ArticleDOI
Added relief in the treatment of acute recurrent sinusitis with adjunctive mometasone furoate nasal spray
Eli O. Meltzer,B.Lauren Charous,William W. Busse,S. James Zinreich,Richard R. Lorber,Melvyn R. Danzig +5 more
TL;DR: The addition of intranasal corticosteroid, MFNS 400 microg twice daily, to antibiotics significantly reduces symptoms of acute sinusitis compared with antibiotic treatment alone.
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Early life origins of asthma
TL;DR: An enhanced understanding of the pathogenesis of childhood asthma will provide the types of insights that will allow us to think in terms of primary disease prevention rather than the present goal of secondary symptom amelioration.
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Relationship of viral infections to wheezing illnesses and asthma.
James E. Gern,William W. Busse +1 more
TL;DR: The influence of viral infections on mechanisms of virus-induced airway inflammation in relationship to the development, persistence and severity of asthma is discussed.
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What effect does asthma treatment have on airway remodeling? Current perspectives
TL;DR: A review will evaluate the development of remodeling in asthmatic subjects and the effects of treatment on these processes, and the need to identify novel asthma indices and phenotypes that correlate with remodeling and might specifically respond to new therapies.
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Elevated bronchoalveolar lavage fluid histamine levels in allergic asthmatics are associated with increased airway obstruction.
TL;DR: Asthmatics and patients with allergic rhinitis had significantly higher BAL fluid histamine than did normal subjects and levels of tryptase and histamine in BAL were not correlated with FEV1 percent predicted in the asthmatic group.