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Anindya Dutta

Researcher at University of Virginia

Publications -  265
Citations -  37109

Anindya Dutta is an academic researcher from University of Virginia. The author has contributed to research in topics: DNA replication & Origin recognition complex. The author has an hindex of 82, co-authored 248 publications receiving 33619 citations. Previous affiliations of Anindya Dutta include Rockefeller University & University of Alabama.

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Identification and analysis of functional elements in 1% of the human genome by the ENCODE pilot project

Ewan Birney, +320 more
- 14 Jun 2007 - 
TL;DR: Functional data from multiple, diverse experiments performed on a targeted 1% of the human genome as part of the pilot phase of the ENCODE Project are reported, providing convincing evidence that the genome is pervasively transcribed, such that the majority of its bases can be found in primary transcripts.
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The ENCODE (ENCyclopedia of DNA elements) Project

Elise A. Feingold, +196 more
- 22 Oct 2004 - 
TL;DR: The ENCyclopedia Of DNA Elements (ENCODE) Project is organized as an international consortium of computational and laboratory-based scientists working to develop and apply high-throughput approaches for detecting all sequence elements that confer biological function.
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p21 in cancer: intricate networks and multiple activities

TL;DR: This Review focuses on recent advances in the understanding of the regulation of p21 and its biological functions with emphasis on its p53-independent tumour suppressor activities and paradoxical tumour-promoting activities, and their implications in cancer.
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DNA replication in eukaryotic cells

TL;DR: This review describes the current understanding of the events of initiation of eukaryotic replication factors and how they are coordinated with cell cycle progression and emphasizes recent progress in determining the function of the different replication factors once they have been assembled at the origin.
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The tumor suppressor microRNA let-7 represses the HMGA2 oncogene

TL;DR: The results provide a novel example of suppression of an oncogene by a tumor-suppressive miRNA and suggest that some tumors activate the oncogen through chromosomal translocations that eliminate the onCogene's 3'UTR with the let-7 target sites.