Herbert Budka

TL;DR: Clinical, radiological, and histological findings of a case of cerebellar granule cell neuronopathy (GCN), a JCV‐associated CNS disease, so far unreported amongst patients treated with natalizumab, are reported.

TL;DR: The first evidence of statistically robust genetic associations in sporadic human prion disease that implicate intracellular trafficking and sphingolipid metabolism as molecular causal mechanisms is presented.

TL;DR: Routine national surveillance methods adjusted as population rates allow objective determination of surveillance intensity, which correlates positively with reported incidence for human prion disease, especially sporadic CJD, largely independent of national context.

TL;DR: The following will outline the consensus of the working group for neuropsychiatric brain banking on ethical guidelines for brain banking, clinical diagnostic criteria, the minimal clinical data set of retrospectively analyzed cases as well as neuropathological standard investigations to perform stageing for neurodegenerative disorders in brain tissue.

TL;DR: The authors report the highly unusual development of a glioblastoma multiforme at the site of excision of a medulloblastomas 6 years earlier, which is considered the likely cause for the later tumor development.