Journal ArticleDOI
Cellular defenses against damage from reactive oxygen species
TLDR
Ceruloplasmin should appear as follows: [See PDF]Abstract:
Pages 139–162: Yu. “Cellular Defenses Against Damage From Reactive Oxygen Species.” Page 150: The section 2. Ceruloplasmin should appear as follows: [See PDF]read more
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L-malate reverses oxidative stress and antioxidative defenses in liver and heart of aged rats
TL;DR: Dietary L-malate was found to enhance the antioxidative defense system with an increased activity of antioxidant enzymes, such as superoxide dismutase (SOD) and glutathione peroxidase (GPx) and increased glutATHione (GSH) levels in the liver of aged rats, a phenomenon not observed in the heart of aged Rats.
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Potentiation of cadmium-induced cytotoxicity by sulfur amino acid deprivation through activation of extracellular signal-regulated kinase1/2 (ERK1/2) in conjunction with p38 kinase or c-jun N-terminal kinase (JNK). Complete inhibition of the potentiated toxicity by U0126 an ERK1/2 and p38 kinase inhibitor.
TL;DR: In this article, the effect of decreased cellular glutathione (GSH) content by sulfur amino acid deprivation on cadmium toxicity and identifying the signaling pathways responsible for the cytotoxicity was investigated.
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Ascites Syndrome in Broiler Chickens: A Review on the Aspect of Endogenous and Exogenous Factors Interactions
TL;DR: Preventive management procedures like different lighting schedules, feed restriction protocols and supplementation of feed with antioxidants which reduce the incidence of ascites in broiler chickens are highlighted.
Journal ArticleDOI
Antioxidant and oxidative status in tissues of manganese superoxide dismutase transgenic mice.
TL;DR: In this article, the expression of the human SOD gene did not systematical alter antioxidant systems or adversely affect the redox state of the transgenic mice, and the results also suggest that expression of human sOD gene confers protection against peroxidative damage to membrane lipids and conjugated dienes and protein carbonyls.
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Antioxidant enzyme activities and mitochondrial fatty acids in pulmonary hypertension syndrome (PHS) in broilers.
TL;DR: It appears that GSH recycling enzyme activities are affected by PHS and high VE presumably in response to differences in oxidative stress and that genetic resistance to PHS is associated with an inherently better capability to metabolize oxidants in lung mitochondria.