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Journal ArticleDOI

Cellular defenses against damage from reactive oxygen species

TLDR
Ceruloplasmin should appear as follows: [See PDF]
Abstract
Pages 139–162: Yu. “Cellular Defenses Against Damage From Reactive Oxygen Species.” Page 150: The section 2. Ceruloplasmin should appear as follows: [See PDF]

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Journal ArticleDOI

Carotenoid availability does not limit the capability of nestling kestrels (Falco tinnunculus) to cope with oxidative stress.

TL;DR: The results show that in general, younger nestlings actually have to cope with a high free radical production, and the ability of wild nestling kestrels to coping with oxidative stress is not affected by carotenoid availability.
Journal ArticleDOI

Antioxidant responses, hepatic intermediary metabolism, histology and ultrastructure in Synechogobius hasta exposed to waterborne cadmium.

TL;DR: Waterborne Cd exposure changed hepatic enzymatic activities involved in intermediary metabolism, induced antioxidant responses and increased lipid peroxidation level in the gill, liver and spleen and induced fatty liver for this fish species.
Journal ArticleDOI

A selenium-dependent glutathione peroxidase (Se-GPx) and two glutathione S-transferases (GSTs) from Chinese shrimp (Fenneropenaeus chinensis)

TL;DR: Tissue distribution and response to pathogens for the three genes was investigated by RT-PCR analysis, which showed that the transcript of FcMuGST and FcGPx increased in response to Vibrio anguillarum infection, while FcThetaGST showed little change at the transcript level.
Journal ArticleDOI

Protective effect of triterpenes on calcium oxalate crystal-induced peroxidative changes in experimental urolithiasis.

TL;DR: Both lupeol and betulin were comparable in their ability to restore the thiol status and the antioxidant enzymes like superoxide dismutase, catalase and glutathione peroxidase, and may involve the inhibition of calcium oxalate crystal aggregation and enhancement of the body defence systems.
Journal ArticleDOI

Depletion of liver glutathione potentiates the oxidative stress and decreases nitric oxide synthesis in a rat endotoxin shock model.

TL;DR: Results show that depletion of the liver glutathione increases the oxidative stress and decreases nitric oxide synthesis of LPS-induced shock in rats.
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