Journal ArticleDOI
Cellular defenses against damage from reactive oxygen species
TLDR
Ceruloplasmin should appear as follows: [See PDF]Abstract:
Pages 139–162: Yu. “Cellular Defenses Against Damage From Reactive Oxygen Species.” Page 150: The section 2. Ceruloplasmin should appear as follows: [See PDF]read more
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Superoxide dismutase expression in human cumulus oophorus cells
TL;DR: Variations in SOD emphasize the relevance of oxidative stress in the oocyte maturation process and suggest that SOD is a potential biomarker for ART success.
Journal ArticleDOI
Proteomic analysis of differentially expressed proteins in Penaeus vannamei hemocytes upon Taura syndrome virus infection
Phattara-orn Chongsatja,Apichai Bourchookarn,Chu Fang Lo,Visith Thongboonkerd,Chartchai Krittanai +4 more
TL;DR: It is hypothesize that C‐ and N‐terminal hemocyanin fragments may have differential roles in hemocytes, and further investigation of these data may lead to better understanding of the molecular responses of crustacean hemocytes to TSV infection.
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Antioxidant role of Umbelliferone in STZ-diabetic rats.
TL;DR: Since UMB treatment decreases lipid peroxidation markers and enhances antioxidants' status it can be considered as a potent antioxidant.
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Acute effects of 1-octyl-3-methylimidazolium bromide ionic liquid on the antioxidant enzyme system of mouse liver.
TL;DR: The results showed that the acute exposure of aqueous ionic liquid could cause damage to mouse, leading to antioxidant responses in livers of the treated mice, including an increase in hepatosomatic index and decrease in liver protein content.
Journal ArticleDOI
Free radical-mediated damage to brain in Alzheimer's disease and its transgenic mouse models.
Joshua A. Sonnen,Joshua A. Sonnen,Joshua A. Sonnen,John C.S. Breitner,John C.S. Breitner,John C.S. Breitner,Mark A. Lovell,Mark A. Lovell,Mark A. Lovell,William R. Markesbery,William R. Markesbery,William R. Markesbery,Joseph F. Quinn,Joseph F. Quinn,Joseph F. Quinn,Thomas J. Montine,Thomas J. Montine,Thomas J. Montine +17 more
TL;DR: Evidence from transgenic mouse models of AD, autopsy samples, and human biofluids obtained during life paying particular attention to the stage of disease is reviewed, highlighting a role for free radical-mediated injury to brain regions from early stages of this illness.