Journal ArticleDOI
Cellular defenses against damage from reactive oxygen species
TLDR
Ceruloplasmin should appear as follows: [See PDF]Abstract:
Pages 139–162: Yu. “Cellular Defenses Against Damage From Reactive Oxygen Species.” Page 150: The section 2. Ceruloplasmin should appear as follows: [See PDF]read more
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Journal ArticleDOI
Comparison of effect of chitin, chitosan, chitosan oligosaccharide and N-acetyl-d-glucosamine on growth performance, antioxidant defenses and oxidative stress status of Penaeus monodon
TL;DR: Results suggested that dietary intake containing chitin or chitosan could enhance the growth performance of P. monodon and improve its resistance to DO stress.
Journal ArticleDOI
The reduced tolerance of rat fatty liver to ischemia reperfusion is associated with mitochondrial oxidative injury
Paolo Caraceni,Marco Domenicali,Gianluigi Vendemiale,Ignazio Grattagliano,A. M. Pertosa,Bruno Nardo,Antonio Maria Morselli-Labate,Franco Trevisani,Giuseppe Palasciano,Emanuele Altomare,Mauro Bernardi +10 more
TL;DR: Fatty infiltration exacerbates mitochondrial oxidative injury during warm ischemia reperfusion and can alter mitochondrial functions, including key processes for ATP synthesis, thus, contributing to the reduced tolerance to reperfusions injury.
Journal ArticleDOI
Iron injections in mice increase skeletal muscle iron content, induce oxidative stress and reduce exercise performance
Trent F. Reardon,David G. Allen +1 more
TL;DR: Iron accumulation in skeletal muscle may play a significant role in the reduced exercise capacity seen in iron overload disorders and in ageing, and may play an underlying role in skeletal Muscle atrophy.
Journal ArticleDOI
Physiological responses of swimming crab Portunus trituberculatus under cold acclimation: Antioxidant defense and heat shock proteins
TL;DR: At low temperatures antioxidant defense system and heat shock proteins in swimming crabs are enhanced to prevent cellular damage and can effectively protect cells against apoptosis; however, cold stress beyond a critical level results in depression of the protective mechanisms, which in turn leads to cellular components damage, and elevated levels of apoptotic cell death.
Book ChapterDOI
The Biological Effects and Possible Modes of Action of Nanosilver
TL;DR: Results of in vitro studies indicate a dose-dependent programmed cell death included by oxidative stress as main possible pathway of toxicity of nanosilver, and some findings indicated a unique nanoparticles effect.