Journal ArticleDOI
Cellular defenses against damage from reactive oxygen species
TLDR
Ceruloplasmin should appear as follows: [See PDF]Abstract:
Pages 139–162: Yu. “Cellular Defenses Against Damage From Reactive Oxygen Species.” Page 150: The section 2. Ceruloplasmin should appear as follows: [See PDF]read more
Citations
More filters
Journal ArticleDOI
Antioxidant Potential of Momordica Charantia in Ammonium Chloride-Induced Hyperammonemic Rats
TL;DR: Results indicate that MCE exerting the antioxidant potentials and maintaining the cellular integrity of the liver tissue could offer protection against AC-induced hyperammonemia.
Journal ArticleDOI
The influence of electronic, steric and hydrophobic properties of flavonoid compounds in the inhibition of the xanthine oxidase
S. L. Da Silva,S. L. Da Silva,A. B. F. da Silva,Káthia Maria Honório,Sergio Marangoni,Marcos H. Toyama,A. F. da Silva +6 more
TL;DR: In this article, the influence of electronic, steric and hydrophobic properties of some flavonoid compounds in inhibiting the XO activity was carried out by using molecular mechanics (MM+) and semi-empirical (AM1) methods.
Journal ArticleDOI
Vitamin C deficiency in the catfish Clarias gariepinus.
Adham,Hashem,Abu-Shabana,Kamel +3 more
TL;DR: Vitamin C is suggested as a potent antioxidant that offers protection against oxidative damage to various fish tissues as well as functional depression of phagocytic engulfment with increased susceptibility to the deteriorating action of pathogenic agents.
Journal ArticleDOI
The preventive inhibition of chondroitin sulfate against the CCl4-induced oxidative stress of subcellular level.
TL;DR: The study suggests that CS might be a potential scavenger of free radicals in the oxidative stress originated from the lipid peroxidation of the liver cells of CCl4-treated rats.
Journal ArticleDOI
p38-MAPK- and Caspase-3-Mediated Superoxide-Induced Apoptosis of Rat Hepatic Stellate Cells: Reversal by Retinoic Acid
Noor Mohamed Jameel,Chinnasamy Thirunavukkarasu,Tong Wu,Simon C. Watkins,Scott L. Friedman,Chandrashekhar R. Gandhi +5 more
TL;DR: It is suggested that the absence of retinoids render aHSCs susceptible to superoxide‐induced apoptosis via caspase‐3 and p38‐MAPK activation, and the effects of oxidative stress induced in vivo caused apoptosis of aHscs in cirrhotic but not of qH SCs in control rats.