Journal ArticleDOI
Cellular defenses against damage from reactive oxygen species
TLDR
Ceruloplasmin should appear as follows: [See PDF]Abstract:
Pages 139–162: Yu. “Cellular Defenses Against Damage From Reactive Oxygen Species.” Page 150: The section 2. Ceruloplasmin should appear as follows: [See PDF]read more
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Differential response of cellular antioxidant mechanism of liver and kidney to arsenic exposure and its relation to dietary protein deficiency
TL;DR: It appears that low-protein diet influences the response of some of the cellular protective components against arsenic insult but does not lead to unique findings.
Journal ArticleDOI
Comparison of the sensitivity of different toxicity test endpoints in a microalga exposed to the herbicide paraquat
Raquel Prado,Rosa María Méndez García,Carmen Rioboo,Concepción Herrero,Julio Abalde,Ángeles Cid +5 more
TL;DR: Taking into account the results obtained, comet assay provides a sensitive and rapid system for measuring primary DNA damage in Chlamydomonas moewusii, which could be an important aspect of environmental genotoxicity monitoring in surface waters.
Journal ArticleDOI
Versatile Fluorescent Probes for Imaging the Superoxide Anion in Living Cells and In Vivo
TL;DR: This minireview systematically summarize the methods for design and application of O2•--responsive fluorescent probes according to the intrinsic characteristic of O 2•-.
Journal ArticleDOI
Changes in Expression of Antioxidant Enzymes Affect Cell-Mediated LDL Oxidation and Oxidized LDL–Induced Apoptosis in Mouse Aortic Cells
Zhong Mao Guo,Zhong Mao Guo,Holly Van Remmen,Holly Van Remmen,Hong Yang,Xin Lian Chen,James Mele,Jan Vijg,Charles J. Epstein,Ye-Shih Ho,Arlan Richardson,Arlan Richardson +11 more
TL;DR: It is suggested that overexpressing Cu/Zn superoxide dismutase or catalase reduces cell-mediated LDL oxidation and oxLDL-induced apoptosis, whereas underexpression of manganesesuperoxide dismUTase or glutathione peroxidase-1 increases cell- mediated LDL oxidation
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Involvement of NADPH oxidase-mediated generation of reactive oxygen species in the apototic cell death by capsaicin in HepG2 human hepatoma cells.
TL;DR: The results suggest that the NADPH oxidase-mediated generation of ROS may be essentially involved in the mechanism of capsaicin-induced apoptosis in HepG2 cells, and suggest that Capsicum may be a valuable agent for the therapeutic intervention of human hepatomas.