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Diseases of swine.
TLDR
In this article, the characteristics of a variety of diseases of swine and methods for their prevention and treatment are described, as well as methods to detect and treat these diseases in swine.Abstract:
Describes the characteristics of a variety of diseases of swine, and methods for their prevention and treatment.read more
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Journal ArticleDOI
Heat-killed Streptococcus suis capsular type 2 strains stimulate tumor necrosis factor alpha and interleukin-6 production by murine macrophages.
TL;DR: The ability of whole killed S. suis type 2 organisms to trigger macrophages to produce proinflammatory cytokines may have an important role in the initiation and development of meningitis caused by this microorganism.
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Lineage and Virulence of Streptococcus suis Serotype 2 Isolates from North America
Nahuel Fittipaldi,Jiangu Xu,Sonia Lacouture,Prasit Tharavichitkul,Makoto Osaki,Tsutomu Sekizaki,Daisuke Takamatsu,Marcelo Gottschalk +7 more
TL;DR: Two sequence types predominate and have lower virulence than other types and can be traced back to E.coli A.
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The pathogenesis of necrotic proliferative colitis in swine is linked to whipworm induced suppression of mucosal immunity to resident bacteria
TL;DR: The complex pathogenesis of necrotic proliferative colitis in pigs may be linked to worm induced suppression of mucosal immunity to resident bacteria and the association between bacteria,lymphocytes and macrophages in the LGCs of pigs infected with T. suis suggests an antigen-processing role for these structures in the colon.
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Isolation of Mycoplasma hyopneumoniae from different sampling sites in experimentally infected and contact SPF piglets.
TL;DR: Results showed that both experimentally infected pigs and contact pigs developed enzootic pneumonia, whatever the route of infection and the strain tested, and suggested that tracheobronchiolar washings could have an influence on the lesion extent observed at necropsy.
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Comparison of the Pathogenesis of Two Genetically Different H3N2 Influenza A Viruses in Pigs
TL;DR: Compared the pathogenicities and replication kinetics of the wholly human virus and a triple-reassortant virus in 7-week-old pigs that were infected intranasally with 2 × 103 to 2 × 106 50% tissue culture infective doses of virus, it is demonstrated that the whollyHuman H3N2 virus replicated to significantly lower titers and that the onset of virus shedding was delayed compared to the replication titers.