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Bernard Mazoyer

Researcher at University of Bordeaux

Publications -  340
Citations -  43021

Bernard Mazoyer is an academic researcher from University of Bordeaux. The author has contributed to research in topics: Population & Hyperintensity. The author has an hindex of 89, co-authored 337 publications receiving 38120 citations. Previous affiliations of Bernard Mazoyer include University of California, Berkeley & French Alternative Energies and Atomic Energy Commission.

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Journal ArticleDOI

Abnormalities of the airways and lung parenchyma in asthmatics: CT observations in 50 patients and inter- and intraobserver variability.

TL;DR: It is concluded that asthmatic patients may exhibit bronchial wall thickening, bronchiectasis and morphological abnormalities suggestive of distal airways disease that can be assessed on CT scans with a clinically acceptable observer variability.
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Human subcortical brain asymmetries in 15,847 people worldwide reveal effects of age and sex

Tulio Guadalupe, +189 more
TL;DR: The largest ever analysis of subcortical brain asymmetries is presented, in a harmonized multi-site study using meta-analysis methods, which revealed that additive genetic factors influenced the asymmetry of these two structures and that of the hippocampus and thalamus.
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Neuroimaging correlates of subjective memory deficits in a community population

TL;DR: Subjective memory deficit (SMD) was associated with neuroimaging characteristics in the temporal and hippocampal regions, suggesting that SMD may, at least in some cases, represent a realistic appraisal of underlying brain function independent of measured cognition.
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Large-vessel correlates of cerebral small-vessel disease

TL;DR: Investigation of the relationship of carotid structure and function with MRI markers of cerebral ischemic small-vessel disease found increasing prevalence of lacunar infarcts and increasing WMHV were associated with increasing prevalence with vascular risk factors.
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Multiple indices of diffusion identifies white matter damage in mild cognitive impairment and Alzheimer's disease.

TL;DR: The findings suggest a scheme where WM damage begins in the core memory network of the temporal lobe, cingulum and prefrontal regions, and spreads beyond these regions in later stages, and DA and MD indices were most sensitive at detecting early changes in MCIa.