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Wei Wen

Researcher at University of New South Wales

Publications -  480
Citations -  21226

Wei Wen is an academic researcher from University of New South Wales. The author has contributed to research in topics: Hyperintensity & Medicine. The author has an hindex of 68, co-authored 445 publications receiving 16724 citations. Previous affiliations of Wei Wen include Nanjing Medical University & University Hospital Bonn.

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Recent advances in electrochemical sensing for hydrogen peroxide: a review

TL;DR: In this article, the electrocatalytic H(2)O( 2) determinations are mainly focused on because they can provide a superior sensing performance over non-electrocatalysttic ones.
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Common genetic variants influence human subcortical brain structures.

Derrek P. Hibar, +344 more
- 09 Apr 2015 - 
TL;DR: In this paper, the authors conduct genome-wide association studies of the volumes of seven subcortical regions and the intracranial volume derived from magnetic resonance images of 30,717 individuals from 50 cohorts.
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The ENIGMA Consortium: large-scale collaborative analyses of neuroimaging and genetic data

Paul M. Thompson, +332 more
TL;DR: The ENIGMA Consortium has detected factors that affect the brain that no individual site could detect on its own, and that require larger numbers of subjects than any individual neuroimaging study has currently collected.
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The genetic architecture of the human cerebral cortex

Katrina L. Grasby, +359 more
- 20 Mar 2020 - 
TL;DR: Results support the radial unit hypothesis that different developmental mechanisms promote surface area expansion and increases in thickness and find evidence that brain structure is a key phenotype along the causal pathway that leads from genetic variation to differences in general cognitive function.
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The neuropsychological profile of vascular cognitive impairment in stroke and TIA patients.

TL;DR: The cognitive deficits in VaD and VCI are characterized by disturbance of frontal functions, with less verbal memory impairment, and the brain lesions that best account for these deficits are noninfarct subcortical white matter and gray matter changes due to ischemia.